Melatonin protects mouse granulosa cells against oxidative damage by inhibiting FOXO1-mediated autophagy: Implication of an antioxidation-independent mechanism

Oxidative stress has been described as a prime driver of granulosa cell (GCs) death during follicular atresia. Increasing evidence suggests potential roles of melatonin in protecting GCs from oxidative injury, though the underlying mechanisms remain largely undetermined. Here we first proposed that...

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Detalles Bibliográficos
Autores principales: Shen, Ming, Cao, Yan, Jiang, Yi, Wei, Yinghui, Liu, Honglin
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2018
Materias:
Research Paper
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6068202/
https://www.ncbi.nlm.nih.gov/pubmed/30014903
http://dx.doi.org/10.1016/j.redox.2018.07.004

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6068202/
https://www.ncbi.nlm.nih.gov/pubmed/30014903
http://dx.doi.org/10.1016/j.redox.2018.07.004

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