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Rapid Brain Death following Cardiac Arrest without Intracranial Pressure Rise and Cerebral Circulation Arrest

We describe here an unusual case of brain death following cardiac arrest. Brain electric activity had totally ceased, allowing the confirmation of brain death, despite normal cerebral blood flow (assessed by both transcranial doppler and tomodensitometry) and no evidence of intracranial hypertension...

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Detalles Bibliográficos
Autores principales: Nguyen, Maxime, Bièvre, Thomas, Nadji, Abdelouaid, Bouhemad, Bélaïd
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Hindawi 2018
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6077554/
https://www.ncbi.nlm.nih.gov/pubmed/30112219
http://dx.doi.org/10.1155/2018/2709174
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author Nguyen, Maxime
Bièvre, Thomas
Nadji, Abdelouaid
Bouhemad, Bélaïd
author_facet Nguyen, Maxime
Bièvre, Thomas
Nadji, Abdelouaid
Bouhemad, Bélaïd
author_sort Nguyen, Maxime
collection PubMed
description We describe here an unusual case of brain death following cardiac arrest. Brain electric activity had totally ceased, allowing the confirmation of brain death, despite normal cerebral blood flow (assessed by both transcranial doppler and tomodensitometry) and no evidence of intracranial hypertension. In our case, a residual electric activity was assessed at admission and lesions worsened on imaging during ICU stay, suggesting that part of the neuronal damage occurred after brain reperfusion. All these elements suggest BD rather by cellular toxicity than intracranial pressure elevation.
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spelling pubmed-60775542018-08-15 Rapid Brain Death following Cardiac Arrest without Intracranial Pressure Rise and Cerebral Circulation Arrest Nguyen, Maxime Bièvre, Thomas Nadji, Abdelouaid Bouhemad, Bélaïd Case Rep Crit Care Case Report We describe here an unusual case of brain death following cardiac arrest. Brain electric activity had totally ceased, allowing the confirmation of brain death, despite normal cerebral blood flow (assessed by both transcranial doppler and tomodensitometry) and no evidence of intracranial hypertension. In our case, a residual electric activity was assessed at admission and lesions worsened on imaging during ICU stay, suggesting that part of the neuronal damage occurred after brain reperfusion. All these elements suggest BD rather by cellular toxicity than intracranial pressure elevation. Hindawi 2018-07-19 /pmc/articles/PMC6077554/ /pubmed/30112219 http://dx.doi.org/10.1155/2018/2709174 Text en Copyright © 2018 Maxime Nguyen et al. https://creativecommons.org/licenses/by/4.0/ This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Case Report
Nguyen, Maxime
Bièvre, Thomas
Nadji, Abdelouaid
Bouhemad, Bélaïd
Rapid Brain Death following Cardiac Arrest without Intracranial Pressure Rise and Cerebral Circulation Arrest
title Rapid Brain Death following Cardiac Arrest without Intracranial Pressure Rise and Cerebral Circulation Arrest
title_full Rapid Brain Death following Cardiac Arrest without Intracranial Pressure Rise and Cerebral Circulation Arrest
title_fullStr Rapid Brain Death following Cardiac Arrest without Intracranial Pressure Rise and Cerebral Circulation Arrest
title_full_unstemmed Rapid Brain Death following Cardiac Arrest without Intracranial Pressure Rise and Cerebral Circulation Arrest
title_short Rapid Brain Death following Cardiac Arrest without Intracranial Pressure Rise and Cerebral Circulation Arrest
title_sort rapid brain death following cardiac arrest without intracranial pressure rise and cerebral circulation arrest
topic Case Report
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6077554/
https://www.ncbi.nlm.nih.gov/pubmed/30112219
http://dx.doi.org/10.1155/2018/2709174
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