Cargando…
Fatty Acids Prevent Hypoxia-Inducible Factor-1α Signaling Through Decreased Succinate in Diabetes
Hypoxia-inducible factor (HIF)-1α is essential following a myocardial infarction (MI), and diabetic patients have poorer prognosis post-MI. Could HIF-1α activation be abnormal in the diabetic heart, and could metabolism be causing this? Diabetic hearts had decreased HIF-1α protein following ischemia...
Autores principales: | Dodd, Michael S., Sousa Fialho, Maria da Luz, Montes Aparicio, Claudia N., Kerr, Matthew, Timm, Kerstin N., Griffin, Julian L., Luiken, Joost J.F.P., Glatz, Jan F.C., Tyler, Damian J., Heather, Lisa C. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Elsevier
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6115650/ https://www.ncbi.nlm.nih.gov/pubmed/30175272 http://dx.doi.org/10.1016/j.jacbts.2018.04.005 |
Ejemplares similares
-
Inhibition of sarcolemmal FAT/CD36 by sulfo-N-succinimidyl oleate rapidly corrects metabolism and restores function in the diabetic heart following hypoxia/reoxygenation
por: Mansor, Latt S., et al.
Publicado: (2017) -
CD36 (SR-B2) as a Target to Treat Lipid Overload-Induced Cardiac Dysfunction
por: Glatz, Jan F.C., et al.
Publicado: (2020) -
CD36 (SR-B2) as master regulator of cellular fatty acid homeostasis
por: Glatz, Jan F.C., et al.
Publicado: (2022) -
Re-balancing cellular energy substrate metabolism to mend the failing heart(☆)
por: Glatz, Jan F.C., et al.
Publicado: (2019) -
Augmenting Vacuolar H(+)-ATPase Function Prevents Cardiomyocytes from Lipid-Overload Induced Dysfunction
por: Wang, Shujin, et al.
Publicado: (2020)