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Intraspinal Anomalies in Early Onset Scoliosis: Current Concepts
Early onset scoliosis (EOS) is deformity of the spine below the age of 5 years. Children with EOS are at risk of impaired thoracic cage development and pulmonary maturation. Initial evaluation consists of determining the etiological cause for EOS, i.e., congenital, neurogenic, idiopathic, or syndrom...
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Formato: | Online Artículo Texto |
Lenguaje: | English |
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Medknow Publications & Media Pvt Ltd
2018
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Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6144598/ https://www.ncbi.nlm.nih.gov/pubmed/30271460 http://dx.doi.org/10.4103/JPN.JPN_100_17 |
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author | Singrakhia, Manoj Malewar, Nikhil Jangle, Ajit |
author_facet | Singrakhia, Manoj Malewar, Nikhil Jangle, Ajit |
author_sort | Singrakhia, Manoj |
collection | PubMed |
description | Early onset scoliosis (EOS) is deformity of the spine below the age of 5 years. Children with EOS are at risk of impaired thoracic cage development and pulmonary maturation. Initial evaluation consists of determining the etiological cause for EOS, i.e., congenital, neurogenic, idiopathic, or syndromic. The advent of magnetic resonance imaging in recent times has lead to increased awareness of neurogenic causes leading to EOS. Evaluation of spinal cord anomalies in EOS is very important as early diagnosis and treatment can help in deformity stabilization and regression. Also, any surgical or nonsurgical intervention to correct the deformity without prior treatment of spinal cord anomalies can lead to disastrous neurological complications. |
format | Online Article Text |
id | pubmed-6144598 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2018 |
publisher | Medknow Publications & Media Pvt Ltd |
record_format | MEDLINE/PubMed |
spelling | pubmed-61445982018-09-28 Intraspinal Anomalies in Early Onset Scoliosis: Current Concepts Singrakhia, Manoj Malewar, Nikhil Jangle, Ajit J Pediatr Neurosci Review Article Early onset scoliosis (EOS) is deformity of the spine below the age of 5 years. Children with EOS are at risk of impaired thoracic cage development and pulmonary maturation. Initial evaluation consists of determining the etiological cause for EOS, i.e., congenital, neurogenic, idiopathic, or syndromic. The advent of magnetic resonance imaging in recent times has lead to increased awareness of neurogenic causes leading to EOS. Evaluation of spinal cord anomalies in EOS is very important as early diagnosis and treatment can help in deformity stabilization and regression. Also, any surgical or nonsurgical intervention to correct the deformity without prior treatment of spinal cord anomalies can lead to disastrous neurological complications. Medknow Publications & Media Pvt Ltd 2018 /pmc/articles/PMC6144598/ /pubmed/30271460 http://dx.doi.org/10.4103/JPN.JPN_100_17 Text en Copyright: © 2018 Journal of Pediatric Neurosciences http://creativecommons.org/licenses/by-nc-sa/4.0 This is an open access journal, and articles are distributed under the terms of the Creative Commons Attribution-NonCommercial-ShareAlike 4.0 License, which allows others to remix, tweak, and build upon the work non-commercially, as long as appropriate credit is given and the new creations are licensed under the identical terms. |
spellingShingle | Review Article Singrakhia, Manoj Malewar, Nikhil Jangle, Ajit Intraspinal Anomalies in Early Onset Scoliosis: Current Concepts |
title | Intraspinal Anomalies in Early Onset Scoliosis: Current Concepts |
title_full | Intraspinal Anomalies in Early Onset Scoliosis: Current Concepts |
title_fullStr | Intraspinal Anomalies in Early Onset Scoliosis: Current Concepts |
title_full_unstemmed | Intraspinal Anomalies in Early Onset Scoliosis: Current Concepts |
title_short | Intraspinal Anomalies in Early Onset Scoliosis: Current Concepts |
title_sort | intraspinal anomalies in early onset scoliosis: current concepts |
topic | Review Article |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6144598/ https://www.ncbi.nlm.nih.gov/pubmed/30271460 http://dx.doi.org/10.4103/JPN.JPN_100_17 |
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