Cargando…
Experimental Traumatic Brain Injury Identifies Distinct Early and Late Phase Axonal Conduction Deficits of White Matter Pathophysiology, and Reveals Intervening Recovery
Traumatic brain injury (TBI) patients often exhibit slowed information processing speed that can underlie diverse symptoms. Processing speed depends on neural circuit function at synapses, in the soma, and along axons. Long axons in white matter (WM) tracts are particularly vulnerable to TBI. We hyp...
Autores principales: | Marion, Christina M., Radomski, Kryslaine L., Cramer, Nathan P., Galdzicki, Zygmunt, Armstrong, Regina C. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Society for Neuroscience
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6181309/ https://www.ncbi.nlm.nih.gov/pubmed/30143572 http://dx.doi.org/10.1523/JNEUROSCI.0819-18.2018 |
Ejemplares similares
-
Acute axon damage and demyelination are mitigated by 4-aminopyridine (4-AP) therapy after experimental traumatic brain injury
por: Radomski, Kryslaine L., et al.
Publicado: (2022) -
Repetitive Blast Exposure Produces White Matter Axon Damage without Subsequent Myelin Remodeling: In Vivo Analysis of Brain Injury Using Fluorescent Reporter Mice
por: Bradshaw, Donald V., et al.
Publicado: (2021) -
Genetic inactivation of SARM1 axon degeneration pathway improves outcome trajectory after experimental traumatic brain injury based on pathological, radiological, and functional measures
por: Bradshaw, Donald V., et al.
Publicado: (2021) -
Repetitive Model of Mild Traumatic Brain Injury Produces Cortical Abnormalities Detectable by Magnetic Resonance Diffusion Imaging, Histopathology, and Behavior
por: Yu, Fengshan, et al.
Publicado: (2017) -
From Abnormal Hippocampal Synaptic Plasticity in Down Syndrome Mouse Models to Cognitive Disability in Down Syndrome
por: Cramer, Nathan, et al.
Publicado: (2012)