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mTOR-dependent autophagy contributes to end-organ resistance and serves as target for treatment in autoimmune disease

Detalles Bibliográficos
Autor principal: Perl, Andras
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2018
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6197622/
https://www.ncbi.nlm.nih.gov/pubmed/30262258
http://dx.doi.org/10.1016/j.ebiom.2018.09.033
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author Perl, Andras
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spelling pubmed-61976222018-10-24 mTOR-dependent autophagy contributes to end-organ resistance and serves as target for treatment in autoimmune disease Perl, Andras EBioMedicine Commentary Elsevier 2018-09-24 /pmc/articles/PMC6197622/ /pubmed/30262258 http://dx.doi.org/10.1016/j.ebiom.2018.09.033 Text en © 2018 The Author http://creativecommons.org/licenses/by-nc-nd/4.0/ This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).
spellingShingle Commentary
Perl, Andras
mTOR-dependent autophagy contributes to end-organ resistance and serves as target for treatment in autoimmune disease
title mTOR-dependent autophagy contributes to end-organ resistance and serves as target for treatment in autoimmune disease
title_full mTOR-dependent autophagy contributes to end-organ resistance and serves as target for treatment in autoimmune disease
title_fullStr mTOR-dependent autophagy contributes to end-organ resistance and serves as target for treatment in autoimmune disease
title_full_unstemmed mTOR-dependent autophagy contributes to end-organ resistance and serves as target for treatment in autoimmune disease
title_short mTOR-dependent autophagy contributes to end-organ resistance and serves as target for treatment in autoimmune disease
title_sort mtor-dependent autophagy contributes to end-organ resistance and serves as target for treatment in autoimmune disease
topic Commentary
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6197622/
https://www.ncbi.nlm.nih.gov/pubmed/30262258
http://dx.doi.org/10.1016/j.ebiom.2018.09.033
work_keys_str_mv AT perlandras mtordependentautophagycontributestoendorganresistanceandservesastargetfortreatmentinautoimmunedisease