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Inflammation as a central mechanism in Alzheimer's disease

Alzheimer's disease (AD) is a progressive neurodegenerative disorder that is characterized by cognitive decline and the presence of two core pathologies, amyloid β plaques and neurofibrillary tangles. Over the last decade, the presence of a sustained immune response in the brain has emerged as...

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Autores principales: Kinney, Jefferson W., Bemiller, Shane M., Murtishaw, Andrew S., Leisgang, Amanda M., Salazar, Arnold M., Lamb, Bruce T.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2018
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6214864/
https://www.ncbi.nlm.nih.gov/pubmed/30406177
http://dx.doi.org/10.1016/j.trci.2018.06.014
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author Kinney, Jefferson W.
Bemiller, Shane M.
Murtishaw, Andrew S.
Leisgang, Amanda M.
Salazar, Arnold M.
Lamb, Bruce T.
author_facet Kinney, Jefferson W.
Bemiller, Shane M.
Murtishaw, Andrew S.
Leisgang, Amanda M.
Salazar, Arnold M.
Lamb, Bruce T.
author_sort Kinney, Jefferson W.
collection PubMed
description Alzheimer's disease (AD) is a progressive neurodegenerative disorder that is characterized by cognitive decline and the presence of two core pathologies, amyloid β plaques and neurofibrillary tangles. Over the last decade, the presence of a sustained immune response in the brain has emerged as a third core pathology in AD. The sustained activation of the brain's resident macrophages (microglia) and other immune cells has been demonstrated to exacerbate both amyloid and tau pathology and may serve as a link in the pathogenesis of the disorder. In the following review, we provide an overview of inflammation in AD and a detailed coverage of a number of microglia-related signaling mechanisms that have been implicated in AD. Additional information on microglia signaling and a number of cytokines in AD are also reviewed. We also review the potential connection of risk factors for AD and how they may be related to inflammatory mechanisms.
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spelling pubmed-62148642018-11-07 Inflammation as a central mechanism in Alzheimer's disease Kinney, Jefferson W. Bemiller, Shane M. Murtishaw, Andrew S. Leisgang, Amanda M. Salazar, Arnold M. Lamb, Bruce T. Alzheimers Dement (N Y) Special Issue from the Centers of Biomedical Research Excellence (COBRE) and Center for Neurodegeneration and Translational Neuroscience (CNTN) Alzheimer's disease (AD) is a progressive neurodegenerative disorder that is characterized by cognitive decline and the presence of two core pathologies, amyloid β plaques and neurofibrillary tangles. Over the last decade, the presence of a sustained immune response in the brain has emerged as a third core pathology in AD. The sustained activation of the brain's resident macrophages (microglia) and other immune cells has been demonstrated to exacerbate both amyloid and tau pathology and may serve as a link in the pathogenesis of the disorder. In the following review, we provide an overview of inflammation in AD and a detailed coverage of a number of microglia-related signaling mechanisms that have been implicated in AD. Additional information on microglia signaling and a number of cytokines in AD are also reviewed. We also review the potential connection of risk factors for AD and how they may be related to inflammatory mechanisms. Elsevier 2018-09-06 /pmc/articles/PMC6214864/ /pubmed/30406177 http://dx.doi.org/10.1016/j.trci.2018.06.014 Text en ©?2018 Published by Elsevier Inc. on behalf of the Alzheimer's Association. http://creativecommons.org/licenses/by-nc-nd/4.0/ This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).
spellingShingle Special Issue from the Centers of Biomedical Research Excellence (COBRE) and Center for Neurodegeneration and Translational Neuroscience (CNTN)
Kinney, Jefferson W.
Bemiller, Shane M.
Murtishaw, Andrew S.
Leisgang, Amanda M.
Salazar, Arnold M.
Lamb, Bruce T.
Inflammation as a central mechanism in Alzheimer's disease
title Inflammation as a central mechanism in Alzheimer's disease
title_full Inflammation as a central mechanism in Alzheimer's disease
title_fullStr Inflammation as a central mechanism in Alzheimer's disease
title_full_unstemmed Inflammation as a central mechanism in Alzheimer's disease
title_short Inflammation as a central mechanism in Alzheimer's disease
title_sort inflammation as a central mechanism in alzheimer's disease
topic Special Issue from the Centers of Biomedical Research Excellence (COBRE) and Center for Neurodegeneration and Translational Neuroscience (CNTN)
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6214864/
https://www.ncbi.nlm.nih.gov/pubmed/30406177
http://dx.doi.org/10.1016/j.trci.2018.06.014
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