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Collective antiskyrmion-mediated phase transition and defect-induced melting in chiral magnetic films
Magnetic phase transitions are a manifestation of competing interactions whose behavior is critically modified by defects and becomes even more complex when topological constraints are involved. In particular, the investigation of skyrmions and skyrmion lattices offers insight into fundamental proce...
Autores principales: | , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2018
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6232090/ https://www.ncbi.nlm.nih.gov/pubmed/30420698 http://dx.doi.org/10.1038/s41598-018-34526-0 |
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author | Pierobon, L. Moutafis, C. Li, Y. Löffler, J. F. Charilaou, M. |
author_facet | Pierobon, L. Moutafis, C. Li, Y. Löffler, J. F. Charilaou, M. |
author_sort | Pierobon, L. |
collection | PubMed |
description | Magnetic phase transitions are a manifestation of competing interactions whose behavior is critically modified by defects and becomes even more complex when topological constraints are involved. In particular, the investigation of skyrmions and skyrmion lattices offers insight into fundamental processes of topological-charge creation and annihilation upon changing the magnetic state. Nonetheless, the exact physical mechanisms behind these phase transitions remain unresolved. Here, we show numerically that it is possible to collectively reverse the polarity of a skyrmion lattice in a field-induced first-order phase transition via a transient antiskyrmion-lattice state. We thus propose a new type of phase transformation where a skyrmion lattice inverts to another one due to topological constraints. In the presence of even a single defect, the process becomes a second-order phase transition with gradual topological-charge melting. This radical change in the system’s behavior from a first-order to a second-order phase transition demonstrates that defects in real materials could prevent us from observing collective topological phenomena. We have systematically compared ultra-thin films with isotropic and anisotropic Dzyaloshinskii-Moriya interactions (DMIs), and demonstrated a nearly identical behavior for such technologically relevant interfacial systems. |
format | Online Article Text |
id | pubmed-6232090 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2018 |
publisher | Nature Publishing Group UK |
record_format | MEDLINE/PubMed |
spelling | pubmed-62320902018-11-28 Collective antiskyrmion-mediated phase transition and defect-induced melting in chiral magnetic films Pierobon, L. Moutafis, C. Li, Y. Löffler, J. F. Charilaou, M. Sci Rep Article Magnetic phase transitions are a manifestation of competing interactions whose behavior is critically modified by defects and becomes even more complex when topological constraints are involved. In particular, the investigation of skyrmions and skyrmion lattices offers insight into fundamental processes of topological-charge creation and annihilation upon changing the magnetic state. Nonetheless, the exact physical mechanisms behind these phase transitions remain unresolved. Here, we show numerically that it is possible to collectively reverse the polarity of a skyrmion lattice in a field-induced first-order phase transition via a transient antiskyrmion-lattice state. We thus propose a new type of phase transformation where a skyrmion lattice inverts to another one due to topological constraints. In the presence of even a single defect, the process becomes a second-order phase transition with gradual topological-charge melting. This radical change in the system’s behavior from a first-order to a second-order phase transition demonstrates that defects in real materials could prevent us from observing collective topological phenomena. We have systematically compared ultra-thin films with isotropic and anisotropic Dzyaloshinskii-Moriya interactions (DMIs), and demonstrated a nearly identical behavior for such technologically relevant interfacial systems. Nature Publishing Group UK 2018-11-12 /pmc/articles/PMC6232090/ /pubmed/30420698 http://dx.doi.org/10.1038/s41598-018-34526-0 Text en © The Author(s) 2018 Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/. |
spellingShingle | Article Pierobon, L. Moutafis, C. Li, Y. Löffler, J. F. Charilaou, M. Collective antiskyrmion-mediated phase transition and defect-induced melting in chiral magnetic films |
title | Collective antiskyrmion-mediated phase transition and defect-induced melting in chiral magnetic films |
title_full | Collective antiskyrmion-mediated phase transition and defect-induced melting in chiral magnetic films |
title_fullStr | Collective antiskyrmion-mediated phase transition and defect-induced melting in chiral magnetic films |
title_full_unstemmed | Collective antiskyrmion-mediated phase transition and defect-induced melting in chiral magnetic films |
title_short | Collective antiskyrmion-mediated phase transition and defect-induced melting in chiral magnetic films |
title_sort | collective antiskyrmion-mediated phase transition and defect-induced melting in chiral magnetic films |
topic | Article |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6232090/ https://www.ncbi.nlm.nih.gov/pubmed/30420698 http://dx.doi.org/10.1038/s41598-018-34526-0 |
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