Cargando…
Knockdown of the translocon protein EXP2 in Plasmodium falciparum reduces growth and protein export
Malaria parasites remodel their host erythrocytes to gain nutrients and avoid the immune system. Host erythrocytes are modified by hundreds of effector proteins exported from the parasite into the host cell. Protein export is mediated by the PTEX translocon comprising five core components of which E...
Autores principales: | Charnaud, Sarah C., Kumarasingha, Rasika, Bullen, Hayley E., Crabb, Brendan S., Gilson, Paul R. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6237293/ https://www.ncbi.nlm.nih.gov/pubmed/30439948 http://dx.doi.org/10.1371/journal.pone.0204785 |
Ejemplares similares
-
A common protein export pathway in malaria parasites
por: Crabb, Brendan, et al.
Publicado: (2010) -
Spatial association with PTEX complexes defines regions for effector export into Plasmodium falciparum-infected erythrocytes
por: Riglar, David T., et al.
Publicado: (2013) -
The Plasmodium falciparum parasitophorous vacuole protein P113 interacts with the parasite protein export machinery and maintains normal vacuole architecture
por: Bullen, Hayley E., et al.
Publicado: (2022) -
A revised mechanism for how Plasmodium falciparum recruits and exports proteins into its erythrocytic host cell
por: Gabriela, Mikha, et al.
Publicado: (2022) -
Characterisation of complexes formed by parasite proteins exported into the host cell compartment of Plasmodium falciparum infected red blood cells
por: Jonsdottir, Thorey K., et al.
Publicado: (2021)