Cargando…
IL-33 suppresses GSK-3β activation through an ST2-independent MyD88/TRAF6/RIP/PI3K/Akt pathway
AIMS: The present study was conducted to explore the effect of interleukin-33 (IL-33) on glycogen synthase kinase-3β (GSK-3β) activation involving Tau phosphorylation, a critical causative factor for Alzheimer's disease (AD). MAIN METHODS: Experiments were performed using PC-12 cells. Target pr...
Autor principal: | Nishizaki, Tomoyuki |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Elsevier
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6260469/ https://www.ncbi.nlm.nih.gov/pubmed/30533546 http://dx.doi.org/10.1016/j.heliyon.2018.e00971 |
Ejemplares similares
-
IL-33 Acts to Express Schaffer Collateral/CA1 LTP and Regulate Learning and Memory by Targeting MyD88
por: Nishizaki, Tomoyuki
Publicado: (2017) -
Deletion of FADD in Macrophages and Granulocytes Results in RIP3- and MyD88-Dependent Systemic Inflammation
por: Schock, Suruchi N., et al.
Publicado: (2015) -
BANK1 interacts with TRAF6 and MyD88 in innate immune signaling in B cells
por: Georg, Ina, et al.
Publicado: (2019) -
A Novel Polysaccharide Isolated From Fresh Longan (Dimocarpus longan Lour.) Activates Macrophage via TLR2/4-Mediated PI3/AKT and MyD88/TRAF6 Pathways
por: Lan, Haibo, et al.
Publicado: (2021) -
MyD88 and beyond: a perspective on MyD88-targeted therapeutic approach for modulation of host immunity
por: Saikh, Kamal U.
Publicado: (2021)