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Sustained hip flexion contracture after femoral lengthening in patients with achondroplasia

BACKGROUND: Hip flexion contracture often occurs after femoral lengthening in patients with achondroplasia, but few studies have investigated its development in these patients. The purpose of this study was to analyze sustained hip flexion contracture in achondroplasia patients who underwent femoral...

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Autores principales: Song, Mi Hyun, Lee, Tae-Jin, Song, Jong Hyeop, Song, Hae-Ryong
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2018
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6267876/
https://www.ncbi.nlm.nih.gov/pubmed/30497473
http://dx.doi.org/10.1186/s12891-018-2344-8
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author Song, Mi Hyun
Lee, Tae-Jin
Song, Jong Hyeop
Song, Hae-Ryong
author_facet Song, Mi Hyun
Lee, Tae-Jin
Song, Jong Hyeop
Song, Hae-Ryong
author_sort Song, Mi Hyun
collection PubMed
description BACKGROUND: Hip flexion contracture often occurs after femoral lengthening in patients with achondroplasia, but few studies have investigated its development in these patients. The purpose of this study was to analyze sustained hip flexion contracture in achondroplasia patients who underwent femoral lengthening and to identify contributing factors. METHODS: This study included 34 patients with achondroplasia who underwent femoral lengthening (mean age at operation, 11.1 years). Sustained hip flexion was defined as flexion contracture lasting > 6 months postoperatively despite physiotherapy. Demographic data, spinopelvic parameters (pelvic incidence, pelvic tilt, sacral slope, lumbar lordosis, and sagittal vertical axis), and quantitative assessments of femoral lengthening were investigated. The associations among these factors and the development of sustained hip flexion contracture were assessed. RESULTS: Sustained hip flexion contracture developed in 13 (38%) of 34 achondroplasia patients after femoral lengthening. Eight (62%) of these 13 patients concomitantly exhibited limitation of knee flexion. Excessive femoral lengthening (odds ratio [OR], 1.450; 95% confidence interval [CI], 1.064 to 1.975; p = 0.019) and forward sagittal vertical axis tilt (OR, 1.062; 95% CI, 1.001 to 1.127; p = 0.047) contributed to sustained hip flexion contracture. CONCLUSIONS: Sustained hip flexion contracture frequently occurs after femoral lengthening in achondroplasia patients. Both excessive femoral lengthening and preoperative forward SVA tilt may contribute to the development of sustained hip flexion contracture in these patients.
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spelling pubmed-62678762018-12-05 Sustained hip flexion contracture after femoral lengthening in patients with achondroplasia Song, Mi Hyun Lee, Tae-Jin Song, Jong Hyeop Song, Hae-Ryong BMC Musculoskelet Disord Research Article BACKGROUND: Hip flexion contracture often occurs after femoral lengthening in patients with achondroplasia, but few studies have investigated its development in these patients. The purpose of this study was to analyze sustained hip flexion contracture in achondroplasia patients who underwent femoral lengthening and to identify contributing factors. METHODS: This study included 34 patients with achondroplasia who underwent femoral lengthening (mean age at operation, 11.1 years). Sustained hip flexion was defined as flexion contracture lasting > 6 months postoperatively despite physiotherapy. Demographic data, spinopelvic parameters (pelvic incidence, pelvic tilt, sacral slope, lumbar lordosis, and sagittal vertical axis), and quantitative assessments of femoral lengthening were investigated. The associations among these factors and the development of sustained hip flexion contracture were assessed. RESULTS: Sustained hip flexion contracture developed in 13 (38%) of 34 achondroplasia patients after femoral lengthening. Eight (62%) of these 13 patients concomitantly exhibited limitation of knee flexion. Excessive femoral lengthening (odds ratio [OR], 1.450; 95% confidence interval [CI], 1.064 to 1.975; p = 0.019) and forward sagittal vertical axis tilt (OR, 1.062; 95% CI, 1.001 to 1.127; p = 0.047) contributed to sustained hip flexion contracture. CONCLUSIONS: Sustained hip flexion contracture frequently occurs after femoral lengthening in achondroplasia patients. Both excessive femoral lengthening and preoperative forward SVA tilt may contribute to the development of sustained hip flexion contracture in these patients. BioMed Central 2018-11-29 /pmc/articles/PMC6267876/ /pubmed/30497473 http://dx.doi.org/10.1186/s12891-018-2344-8 Text en © The Author(s). 2018 Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.
spellingShingle Research Article
Song, Mi Hyun
Lee, Tae-Jin
Song, Jong Hyeop
Song, Hae-Ryong
Sustained hip flexion contracture after femoral lengthening in patients with achondroplasia
title Sustained hip flexion contracture after femoral lengthening in patients with achondroplasia
title_full Sustained hip flexion contracture after femoral lengthening in patients with achondroplasia
title_fullStr Sustained hip flexion contracture after femoral lengthening in patients with achondroplasia
title_full_unstemmed Sustained hip flexion contracture after femoral lengthening in patients with achondroplasia
title_short Sustained hip flexion contracture after femoral lengthening in patients with achondroplasia
title_sort sustained hip flexion contracture after femoral lengthening in patients with achondroplasia
topic Research Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6267876/
https://www.ncbi.nlm.nih.gov/pubmed/30497473
http://dx.doi.org/10.1186/s12891-018-2344-8
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