Cargando…
Hippocampal CA1 βCaMKII mediates neuroinflammatory responses via COX-2/PGE2 signaling pathways in depression
BACKGROUND: Neuroinflammation has recently emerged as a critical risk factor in the pathophysiology of depression. However, the underlying molecular mechanisms and the development of novel therapeutic strategies as means to target these inflammatory pathways for use in the treatment of depression re...
Autores principales: | Song, Qiqi, Fan, Cuiqin, Wang, Peng, Li, Ye, Yang, Mu, Yu, Shu Yan |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6286788/ https://www.ncbi.nlm.nih.gov/pubmed/30526621 http://dx.doi.org/10.1186/s12974-018-1377-0 |
Ejemplares similares
-
Does CaMKII decode Ca(2+) oscillations?
por: Pinto, Thiago M, et al.
Publicado: (2012) -
CaMKII points the way
por: Leslie, Mitch
Publicado: (2012) -
CaMKII in Cardiometabolic Disease
por: Ozcan, Lale, et al.
Publicado: (2014) -
Synaptic memory and CaMKII
por: Nicoll, Roger A., et al.
Publicado: (2023) -
The role of hippocampal CaMKII in resilience to trauma-related psychopathology
por: Hazra, Somoday, et al.
Publicado: (2022)