Cargando…
Neuroprotective Effects of Exogenous Activin A on Oxygen-Glucose Deprivation in PC12 Cells
Ischemic cerebrovascular disease is one of the most common causes of death in the World. Exogenous activin A (ActA) protects neurons against toxicity and plays a central role in regulating the brain’s response to injury. In the present study, we investigated the mechanisms involved in the neuroprote...
Autores principales: | He, Jin-Ting, Mang, Jing, Mei, Chun-Li, Yang, Le, Wang, Jiao-Qi, Xing, Ying, Yang, Hong, Xu, Zhong-Xin |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2011
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6290572/ https://www.ncbi.nlm.nih.gov/pubmed/22210170 http://dx.doi.org/10.3390/molecules17010315 |
Ejemplares similares
-
Activin A prevents neuron-like PC12 cell apoptosis after oxygen-glucose deprivation☆
por: Xu, Guihua, et al.
Publicado: (2013) -
Neuroprotective effects of Activin A on endoplasmic reticulum stress-mediated apoptotic and autophagic PC12 cell death
por: Xue, Long-xing, et al.
Publicado: (2017) -
Neuroprotective effects of leonurine against oxygen–glucose deprivation by targeting Cx36/CaMKII in PC12 cells
por: Li, Jiao, et al.
Publicado: (2018) -
Endogenous Protection Derived from Activin A/Smads Transduction Loop Stimulated via Ischemic Injury in PC12 Cells
por: Mang, Jing, et al.
Publicado: (2013) -
Neuroprotective mechanism of low-dose sodium nitrite in oxygen-glucose deprivation model of cerebral ischemic stroke in PC12 cells
por: Shakib, Nader, et al.
Publicado: (2019)