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Epicatechin modulates stress-resistance in C. elegans via insulin/IGF-1 signaling pathway

The nematode Caenorhabditis elegans has been used to examine the influence of epicatechin (EC), an abundant flavonoid in the human diet, in some stress biomarkers (ROS production, lipid peroxidation and protein carbonylation). Furthermore, the ability of EC to modulate the expression of some key gen...

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Autores principales: Ayuda-Durán, Begoña, González-Manzano, Susana, Miranda-Vizuete, Antonio, Dueñas, Montserrat, Santos-Buelga, Celestino, González-Paramás, Ana M.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Public Library of Science 2019
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6349306/
https://www.ncbi.nlm.nih.gov/pubmed/30689636
http://dx.doi.org/10.1371/journal.pone.0199483
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author Ayuda-Durán, Begoña
González-Manzano, Susana
Miranda-Vizuete, Antonio
Dueñas, Montserrat
Santos-Buelga, Celestino
González-Paramás, Ana M.
author_facet Ayuda-Durán, Begoña
González-Manzano, Susana
Miranda-Vizuete, Antonio
Dueñas, Montserrat
Santos-Buelga, Celestino
González-Paramás, Ana M.
author_sort Ayuda-Durán, Begoña
collection PubMed
description The nematode Caenorhabditis elegans has been used to examine the influence of epicatechin (EC), an abundant flavonoid in the human diet, in some stress biomarkers (ROS production, lipid peroxidation and protein carbonylation). Furthermore, the ability of EC to modulate the expression of some key genes in the insulin/IGF-1 signaling pathway (IIS), involved in longevity and oxidative or heat shock stress response, has also been explored. The final aim was to contribute to the elucidation of the mechanisms involved in the biological effects of flavonoids. The results showed that EC-treated wild-type C. elegans exhibited increased survival and reduced oxidative damage of biomolecules when submitted to thermal stress. EC treatment led to a moderate elevation in ROS levels, which might activate endogenous mechanisms of defense protecting against oxidative insult. The enhanced stress resistance induced by EC was found to be mediated through the IIS pathway, since assays in daf-2, age-1, akt-1, akt-2, sgk-1, daf-16, skn-1 and hsf-1 loss of function mutant strains failed to show any heat-resistant phenotype against thermal stress when treated with EC. Consistently, EC treatment upregulated the expression of some stress resistance associated genes, such as gst-4, hsp-16.2 and hsp-70, which are downstream regulated by the IIS pathway.
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spelling pubmed-63493062019-02-15 Epicatechin modulates stress-resistance in C. elegans via insulin/IGF-1 signaling pathway Ayuda-Durán, Begoña González-Manzano, Susana Miranda-Vizuete, Antonio Dueñas, Montserrat Santos-Buelga, Celestino González-Paramás, Ana M. PLoS One Research Article The nematode Caenorhabditis elegans has been used to examine the influence of epicatechin (EC), an abundant flavonoid in the human diet, in some stress biomarkers (ROS production, lipid peroxidation and protein carbonylation). Furthermore, the ability of EC to modulate the expression of some key genes in the insulin/IGF-1 signaling pathway (IIS), involved in longevity and oxidative or heat shock stress response, has also been explored. The final aim was to contribute to the elucidation of the mechanisms involved in the biological effects of flavonoids. The results showed that EC-treated wild-type C. elegans exhibited increased survival and reduced oxidative damage of biomolecules when submitted to thermal stress. EC treatment led to a moderate elevation in ROS levels, which might activate endogenous mechanisms of defense protecting against oxidative insult. The enhanced stress resistance induced by EC was found to be mediated through the IIS pathway, since assays in daf-2, age-1, akt-1, akt-2, sgk-1, daf-16, skn-1 and hsf-1 loss of function mutant strains failed to show any heat-resistant phenotype against thermal stress when treated with EC. Consistently, EC treatment upregulated the expression of some stress resistance associated genes, such as gst-4, hsp-16.2 and hsp-70, which are downstream regulated by the IIS pathway. Public Library of Science 2019-01-28 /pmc/articles/PMC6349306/ /pubmed/30689636 http://dx.doi.org/10.1371/journal.pone.0199483 Text en © 2019 Ayuda-Durán et al http://creativecommons.org/licenses/by/4.0/ This is an open access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/) , which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
spellingShingle Research Article
Ayuda-Durán, Begoña
González-Manzano, Susana
Miranda-Vizuete, Antonio
Dueñas, Montserrat
Santos-Buelga, Celestino
González-Paramás, Ana M.
Epicatechin modulates stress-resistance in C. elegans via insulin/IGF-1 signaling pathway
title Epicatechin modulates stress-resistance in C. elegans via insulin/IGF-1 signaling pathway
title_full Epicatechin modulates stress-resistance in C. elegans via insulin/IGF-1 signaling pathway
title_fullStr Epicatechin modulates stress-resistance in C. elegans via insulin/IGF-1 signaling pathway
title_full_unstemmed Epicatechin modulates stress-resistance in C. elegans via insulin/IGF-1 signaling pathway
title_short Epicatechin modulates stress-resistance in C. elegans via insulin/IGF-1 signaling pathway
title_sort epicatechin modulates stress-resistance in c. elegans via insulin/igf-1 signaling pathway
topic Research Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6349306/
https://www.ncbi.nlm.nih.gov/pubmed/30689636
http://dx.doi.org/10.1371/journal.pone.0199483
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