Cancer-Associated Intermediate Conductance Ca(2+)-Activated K(+) Channel K(Ca)3.1

Several tumor entities have been reported to overexpress K(Ca)3.1 potassium channels due to epigenetic, transcriptional, or post-translational modifications. By modulating membrane potential, cell volume, or Ca(2+) signaling, K(Ca)3.1 has been proposed to exert pivotal oncogenic functions in tumorig...

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Detalles Bibliográficos
Autores principales: Mohr, Corinna J., Steudel, Friederike A., Gross, Dominic, Ruth, Peter, Lo, Wing-Yee, Hoppe, Reiner, Schroth, Werner, Brauch, Hiltrud, Huber, Stephan M., Lukowski, Robert
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2019
Materias:
Review
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6357066/
https://www.ncbi.nlm.nih.gov/pubmed/30658505
http://dx.doi.org/10.3390/cancers11010109

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6357066/
https://www.ncbi.nlm.nih.gov/pubmed/30658505
http://dx.doi.org/10.3390/cancers11010109

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