Intestinal Lipolysis Mitigates Nonalcoholic Fatty Liver Disease: New Roles for Carboxylesterase 2c in the Intestine

The most important argument of this study is that modulation of intestinal lipid metabolism by enhancing intestinal Ces2c expression has the potential to affect hepatic and systemic lipid metabolism, regulating the development of NAFLD and systemic insulin resistance.

Detalles Bibliográficos
Autores principales: Pimienta, Michael, Beletsky, Alexander, Seki, Ekihiro
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2019
Materias:
Editorial
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6357825/
https://www.ncbi.nlm.nih.gov/pubmed/30766955
http://dx.doi.org/10.1002/hep4.1318
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author Pimienta, Michael
Beletsky, Alexander
Seki, Ekihiro
author_facet Pimienta, Michael
Beletsky, Alexander
Seki, Ekihiro
author_sort Pimienta, Michael
collection PubMed
description The most important argument of this study is that modulation of intestinal lipid metabolism by enhancing intestinal Ces2c expression has the potential to affect hepatic and systemic lipid metabolism, regulating the development of NAFLD and systemic insulin resistance.
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spelling pubmed-63578252019-02-14 Intestinal Lipolysis Mitigates Nonalcoholic Fatty Liver Disease: New Roles for Carboxylesterase 2c in the Intestine Pimienta, Michael Beletsky, Alexander Seki, Ekihiro Hepatol Commun Editorial The most important argument of this study is that modulation of intestinal lipid metabolism by enhancing intestinal Ces2c expression has the potential to affect hepatic and systemic lipid metabolism, regulating the development of NAFLD and systemic insulin resistance. John Wiley and Sons Inc. 2019-02-01 /pmc/articles/PMC6357825/ /pubmed/30766955 http://dx.doi.org/10.1002/hep4.1318 Text en © 2019 The Authors. Hepatology Communications published by Wiley Periodicals, Inc., on behalf of the American Association for the Study of Liver Diseases. This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc-nd/4.0/ License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.
spellingShingle Editorial
Pimienta, Michael
Beletsky, Alexander
Seki, Ekihiro
Intestinal Lipolysis Mitigates Nonalcoholic Fatty Liver Disease: New Roles for Carboxylesterase 2c in the Intestine
title Intestinal Lipolysis Mitigates Nonalcoholic Fatty Liver Disease: New Roles for Carboxylesterase 2c in the Intestine
title_full Intestinal Lipolysis Mitigates Nonalcoholic Fatty Liver Disease: New Roles for Carboxylesterase 2c in the Intestine
title_fullStr Intestinal Lipolysis Mitigates Nonalcoholic Fatty Liver Disease: New Roles for Carboxylesterase 2c in the Intestine
title_full_unstemmed Intestinal Lipolysis Mitigates Nonalcoholic Fatty Liver Disease: New Roles for Carboxylesterase 2c in the Intestine
title_short Intestinal Lipolysis Mitigates Nonalcoholic Fatty Liver Disease: New Roles for Carboxylesterase 2c in the Intestine
title_sort intestinal lipolysis mitigates nonalcoholic fatty liver disease: new roles for carboxylesterase 2c in the intestine
topic Editorial
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6357825/
https://www.ncbi.nlm.nih.gov/pubmed/30766955
http://dx.doi.org/10.1002/hep4.1318
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AT beletskyalexander intestinallipolysismitigatesnonalcoholicfattyliverdiseasenewrolesforcarboxylesterase2cintheintestine
AT sekiekihiro intestinallipolysismitigatesnonalcoholicfattyliverdiseasenewrolesforcarboxylesterase2cintheintestine

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