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Transient complete heart block following catheter ablation of a left lateral accessory pathway

A 16‐year‐old female with symptomatic Wolff‐Parkinson‐White (WPW) syndrome underwent catheter ablation of a left‐sided lateral accessory pathway. The accessory pathway was eliminated with the first ablation lesion; however, the patient immediately developed complete heart block (CHB). At first, comp...

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Autores principales: Schweis, Franz, Ho, Gordon, Krummen, David E., Hoffmayer, Kurt, Birgersdotter‐Green, Ulrika, Feld, Gregory
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2018
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6373828/
https://www.ncbi.nlm.nih.gov/pubmed/30805061
http://dx.doi.org/10.1002/joa3.12138
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author Schweis, Franz
Ho, Gordon
Krummen, David E.
Hoffmayer, Kurt
Birgersdotter‐Green, Ulrika
Feld, Gregory
author_facet Schweis, Franz
Ho, Gordon
Krummen, David E.
Hoffmayer, Kurt
Birgersdotter‐Green, Ulrika
Feld, Gregory
author_sort Schweis, Franz
collection PubMed
description A 16‐year‐old female with symptomatic Wolff‐Parkinson‐White (WPW) syndrome underwent catheter ablation of a left‐sided lateral accessory pathway. The accessory pathway was eliminated with the first ablation lesion; however, the patient immediately developed complete heart block (CHB). At first, complete heart block was thought to be due to ablation of left atrial extension of the AV node, and pacemaker therapy was considered. However, careful ECG analysis revealed that the development of CHB was in fact due to bump injury to the AV node during transseptal catheterization. Conservative management allowed resolution of AV nodal conduction without need for a permanent pacemaker.
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spelling pubmed-63738282019-02-25 Transient complete heart block following catheter ablation of a left lateral accessory pathway Schweis, Franz Ho, Gordon Krummen, David E. Hoffmayer, Kurt Birgersdotter‐Green, Ulrika Feld, Gregory J Arrhythm Case Reports A 16‐year‐old female with symptomatic Wolff‐Parkinson‐White (WPW) syndrome underwent catheter ablation of a left‐sided lateral accessory pathway. The accessory pathway was eliminated with the first ablation lesion; however, the patient immediately developed complete heart block (CHB). At first, complete heart block was thought to be due to ablation of left atrial extension of the AV node, and pacemaker therapy was considered. However, careful ECG analysis revealed that the development of CHB was in fact due to bump injury to the AV node during transseptal catheterization. Conservative management allowed resolution of AV nodal conduction without need for a permanent pacemaker. John Wiley and Sons Inc. 2018-11-03 /pmc/articles/PMC6373828/ /pubmed/30805061 http://dx.doi.org/10.1002/joa3.12138 Text en © 2018 The Authors. Journal of Arrhythmia published by John Wiley & Sons Australia, Ltd on behalf of the Japanese Heart Rhythm Society. This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited and is not used for commercial purposes.
spellingShingle Case Reports
Schweis, Franz
Ho, Gordon
Krummen, David E.
Hoffmayer, Kurt
Birgersdotter‐Green, Ulrika
Feld, Gregory
Transient complete heart block following catheter ablation of a left lateral accessory pathway
title Transient complete heart block following catheter ablation of a left lateral accessory pathway
title_full Transient complete heart block following catheter ablation of a left lateral accessory pathway
title_fullStr Transient complete heart block following catheter ablation of a left lateral accessory pathway
title_full_unstemmed Transient complete heart block following catheter ablation of a left lateral accessory pathway
title_short Transient complete heart block following catheter ablation of a left lateral accessory pathway
title_sort transient complete heart block following catheter ablation of a left lateral accessory pathway
topic Case Reports
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6373828/
https://www.ncbi.nlm.nih.gov/pubmed/30805061
http://dx.doi.org/10.1002/joa3.12138
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