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Unusual FDG-PET Findings in Traumatic Brain Injury; Did Traumatic Brain Injury Provoke Rapid Progression of Alzheimer's Disease?
Traumatic brain injury (TBI) is common, and is often the leading cause of disability and death. Complications after TBI include increased risk for chronic central nervous system disease, such as Alzheimer's disease (AD). However, the pathophysiology relating acute injury to neurodegeneration is...
Autores principales: | , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Korean Dementia Association
2017
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6427956/ https://www.ncbi.nlm.nih.gov/pubmed/30906371 http://dx.doi.org/10.12779/dnd.2017.16.2.54 |
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author | Hwang, Inha Ha, Sang-Won Han, Jeong Ho |
author_facet | Hwang, Inha Ha, Sang-Won Han, Jeong Ho |
author_sort | Hwang, Inha |
collection | PubMed |
description | Traumatic brain injury (TBI) is common, and is often the leading cause of disability and death. Complications after TBI include increased risk for chronic central nervous system disease, such as Alzheimer's disease (AD). However, the pathophysiology relating acute injury to neurodegeneration is unclear. Here we present a case of a patient whose cognition declined after TBI, and whose 18F fluorodeoxyglucose positron emission tomography scan showed an AD pattern. |
format | Online Article Text |
id | pubmed-6427956 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2017 |
publisher | Korean Dementia Association |
record_format | MEDLINE/PubMed |
spelling | pubmed-64279562019-03-22 Unusual FDG-PET Findings in Traumatic Brain Injury; Did Traumatic Brain Injury Provoke Rapid Progression of Alzheimer's Disease? Hwang, Inha Ha, Sang-Won Han, Jeong Ho Dement Neurocogn Disord Letter to the Editor Traumatic brain injury (TBI) is common, and is often the leading cause of disability and death. Complications after TBI include increased risk for chronic central nervous system disease, such as Alzheimer's disease (AD). However, the pathophysiology relating acute injury to neurodegeneration is unclear. Here we present a case of a patient whose cognition declined after TBI, and whose 18F fluorodeoxyglucose positron emission tomography scan showed an AD pattern. Korean Dementia Association 2017-06 2017-06-30 /pmc/articles/PMC6427956/ /pubmed/30906371 http://dx.doi.org/10.12779/dnd.2017.16.2.54 Text en © 2017 Korean Dementia Association http://creativecommons.org/licenses/by-nc/4.0/ This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. |
spellingShingle | Letter to the Editor Hwang, Inha Ha, Sang-Won Han, Jeong Ho Unusual FDG-PET Findings in Traumatic Brain Injury; Did Traumatic Brain Injury Provoke Rapid Progression of Alzheimer's Disease? |
title | Unusual FDG-PET Findings in Traumatic Brain Injury; Did Traumatic Brain Injury Provoke Rapid Progression of Alzheimer's Disease? |
title_full | Unusual FDG-PET Findings in Traumatic Brain Injury; Did Traumatic Brain Injury Provoke Rapid Progression of Alzheimer's Disease? |
title_fullStr | Unusual FDG-PET Findings in Traumatic Brain Injury; Did Traumatic Brain Injury Provoke Rapid Progression of Alzheimer's Disease? |
title_full_unstemmed | Unusual FDG-PET Findings in Traumatic Brain Injury; Did Traumatic Brain Injury Provoke Rapid Progression of Alzheimer's Disease? |
title_short | Unusual FDG-PET Findings in Traumatic Brain Injury; Did Traumatic Brain Injury Provoke Rapid Progression of Alzheimer's Disease? |
title_sort | unusual fdg-pet findings in traumatic brain injury; did traumatic brain injury provoke rapid progression of alzheimer's disease? |
topic | Letter to the Editor |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6427956/ https://www.ncbi.nlm.nih.gov/pubmed/30906371 http://dx.doi.org/10.12779/dnd.2017.16.2.54 |
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