Cargando…
Cardiomyocyte substructure reverts to an immature phenotype during heart failure
KEY POINTS: As reactivation of the fetal gene program has been implicated in pathological remodelling during heart failure (HF), we examined whether cardiomyocyte subcellular structure and function revert to an immature phenotype during this disease. Surface and internal membrane structures appeared...
Autores principales: | Lipsett, D. B., Frisk, M., Aronsen, J. M., Nordén, E. S., Buonarati, O. R., Cataliotti, A., Hell, J. W., Sjaastad, I., Christensen, G., Louch, W. E. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2019
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6441900/ https://www.ncbi.nlm.nih.gov/pubmed/30707448 http://dx.doi.org/10.1113/JP277273 |
Ejemplares similares
-
Elevated ventricular wall stress disrupts cardiomyocyte t-tubule structure and calcium homeostasis
por: Frisk, Michael, et al.
Publicado: (2016) -
Etiology-Dependent Impairment of Diastolic Cardiomyocyte Calcium Homeostasis in Heart Failure With Preserved Ejection Fraction
por: Frisk, Michael, et al.
Publicado: (2021) -
Exercise Training Stabilizes RyR2-Dependent Ca(2+) Release in Post-infarction Heart Failure
por: Danielsen, Tore Kristian, et al.
Publicado: (2021) -
Targeting Cardiomyocyte Ca(2+) Homeostasis in Heart Failure
por: Røe, Åsmund T., et al.
Publicado: (2015) -
Regional diastolic dysfunction in post-infarction heart failure: role of local mechanical load and SERCA expression
por: Røe, Åsmund T, et al.
Publicado: (2019)