Cargando…

Amyloid Network Topology Characterizes the Progression of Alzheimer’s Disease During the Predementia Stages

There is increasing evidence showing that the accumulation of the amyloid-β (Aβ) peptide into extracellular plaques is a central event in Alzheimer's disease (AD). These abnormalities can be detected as lowered levels of Aβ(42) in the cerebrospinal fluid (CSF) and are followed by increased amyl...

Descripción completa

Detalles Bibliográficos
Autores principales:	Pereira, Joana B, Strandberg, Tor Olof, Palmqvist, Sebastian, Volpe, Giovanni, van Westen, Danielle, Westman, Eric, Hansson, Oskar
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	Oxford University Press 2018
Materias:	Original Articles
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6454565/ https://www.ncbi.nlm.nih.gov/pubmed/29136123 http://dx.doi.org/10.1093/cercor/bhx294

Ejemplares similares

Amyloid and tau accumulate across distinct spatial networks and are differentially associated with brain connectivity
por: Pereira, Joana B, et al.
Publicado: (2019)

Association Between Earliest Amyloid Uptake and Functional Connectivity in Cognitively Unimpaired Elderly
por: Hahn, Andreas, et al.
Publicado: (2019)

Brain myoinositol as a potential marker of amyloid-related pathology: A longitudinal study
por: Voevodskaya, Olga, et al.
Publicado: (2019)

Cerebral perfusion in the predementia stages of Alzheimer’s disease
por: Binnewijzend, Maja A. A., et al.
Publicado: (2015)

Medial temporal atrophy in preclinical dementia: Visual and automated assessment during six year follow-up
por: Mårtensson, Gustav, et al.
Publicado: (2020)

Cerebrovascular and amyloid pathology in predementia stages: the relationship with neurodegeneration and cognitive decline
por: Bos, Isabelle, et al.
Publicado: (2017)

Plasma markers predict changes in amyloid, tau, atrophy and cognition in non-demented subjects
por: Pereira, Joana B, et al.
Publicado: (2021)

Cerebral hypoperfusion is not associated with an increase in amyloid β pathology in middle-aged or elderly people
por: Hansson, Oskar, et al.
Publicado: (2018)

Medial temporal lobe connectivity and its associations with cognition in early Alzheimer’s disease
por: Berron, David, et al.
Publicado: (2020)

Corrigendum to: Medial temporal lobe connectivity and its associations with cognition in early Alzheimer’s disease
por: Berron, David, et al.
Publicado: (2021)

Plasma β-amyloid in Alzheimer’s disease and vascular disease
por: Janelidze, Shorena, et al.
Publicado: (2016)

Diagnosis and biomarkers of predementia in Alzheimer's disease
por: Forlenza, Orestes V, et al.
Publicado: (2010)

Gray matter hypoperfusion is a late pathological event in the course of Alzheimer’s disease
por: Ahmadi, Khazar, et al.
Publicado: (2022)

Correction to: Cerebrovascular and amyloid pathology in predementia stages: the relationship with neurodegeneration and cognitive decline
por: Bos, Isabelle, et al.
Publicado: (2018)

Myo-inositol changes precede amyloid pathology and relate to APOE genotype in Alzheimer disease
por: Voevodskaya, Olga, et al.
Publicado: (2016)

A quick test of cognitive speed can predict development of dementia in Parkinson’s disease
por: Jalakas, Mattis, et al.
Publicado: (2019)

Associations of CSF PDGFRβ With Aging, Blood-Brain Barrier Damage, Neuroinflammation, and Alzheimer Disease Pathologic Changes
por: Cicognola, Claudia, et al.
Publicado: (2023)

Relationship between cortical iron and tau aggregation in Alzheimer’s disease
por: Spotorno, Nicola, et al.
Publicado: (2020)

The Effects of Tau, Amyloid, and White Matter Lesions on Mobility, Dual Tasking, and Balance in Older People
por: Nilsson, Maria H, et al.
Publicado: (2020)

Measures of cortical microstructure are linked to amyloid pathology in Alzheimer’s disease
por: Spotorno, Nicola, et al.
Publicado: (2022)

Enrichment and Stratification for Predementia Alzheimer Disease Clinical Trials
por: Holland, Dominic, et al.
Publicado: (2012)

Astrocytic function is associated with both amyloid-β and tau pathology in non-demented APOE ϵ4 carriers
por: Spotorno, Nicola, et al.
Publicado: (2022)

Microglial activation protects against accumulation of tau aggregates in nondemented individuals with underlying Alzheimer’s disease pathology
por: Pereira, Joana B., et al.
Publicado: (2022)

The implications of different approaches to define AT(N) in Alzheimer disease
por: Mattsson-Carlgren, Niklas, et al.
Publicado: (2020)

Associations between tau, Aβ, and cortical thickness with cognition in Alzheimer disease
por: Ossenkoppele, Rik, et al.
Publicado: (2019)

Automatic speech analysis for the assessment of patients with predementia and Alzheimer's disease
por: König, Alexandra, et al.
Publicado: (2015)

Disrupted Network Topology in Patients with Stable and Progressive Mild Cognitive Impairment and Alzheimer's Disease
por: Pereira, Joana B., et al.
Publicado: (2016)

Prefrontal EEG slowing, synchronization, and ERP peak latency in association with predementia stages of Alzheimer’s disease
por: Choi, Jungmi, et al.
Publicado: (2023)

The age-related effect on cognitive performance in cognitively healthy elderly is mainly caused by underlying AD pathology or cerebrovascular lesions: implications for cutoffs regarding cognitive impairment
por: Borland, Emma, et al.
Publicado: (2020)

Plasma GFAP is an early marker of amyloid-β but not tau pathology in Alzheimer’s disease
por: Pereira, Joana B, et al.
Publicado: (2021)

Longitudinal plasma p-tau217 is increased in early stages of Alzheimer’s disease
por: Mattsson-Carlgren, Niklas, et al.
Publicado: (2020)

Nonlinear changes in delayed functional network topology in Alzheimer’s disease: relationship with amyloid and tau pathology
por: Mijalkov, Mite, et al.
Publicado: (2023)

CSF Aβ42/Aβ40 and Aβ42/Aβ38 ratios: better diagnostic markers of Alzheimer disease
por: Janelidze, Shorena, et al.
Publicado: (2016)

β-Amyloid–Dependent and –Independent Genetic Pathways Regulating CSF Tau Biomarkers in Alzheimer Disease
por: Kumar, Atul, et al.
Publicado: (2022)

The accumulation rate of tau aggregates is higher in females and younger amyloid-positive subjects
por: Smith, Ruben, et al.
Publicado: (2020)

Amyloid-associated increases in soluble tau relate to tau aggregation rates and cognitive decline in early Alzheimer’s disease
por: Pichet Binette, Alexa, et al.
Publicado: (2022)

Earliest accumulation of β-amyloid occurs within the default-mode network and concurrently affects brain connectivity
por: Palmqvist, Sebastian, et al.
Publicado: (2017)

Detailed comparison of amyloid PET and CSF biomarkers for identifying early Alzheimer disease
por: Palmqvist, Sebastian, et al.
Publicado: (2015)

Atrophy of the Posterior Subiculum Is Associated with Memory Impairment, Tau- and Aβ Pathology in Non-demented Individuals
por: Lindberg, Olof, et al.
Publicado: (2017)

Plasma amyloid-β42/40 and apolipoprotein E for amyloid PET pre-screening in secondary prevention trials of Alzheimer’s disease
por: Cullen, Nicholas C, et al.
Publicado: (2023)

Cannot write session to /tmp/vufind_sessions/sess_90crp7go69eup3pjtu2fbtludj