Toll-like receptor 3-mediated inflammation by p38 is enhanced by endothelial nitric oxide synthase knockdown

BACKGROUND: Vascular dysfunction is commonly seen during severe viral infections. Endothelial nitric oxide synthase (eNOS), has been postulated to play an important role in regulating vascular homeostasis as well as propagation of the inflammatory reaction. We hypothesized that the loss of eNOS woul...

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Detalles Bibliográficos
Autores principales: Koch, Stephen R., Choi, Hyehun, Mace, Eric H., Stark, Ryan J.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2019
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6466662/
https://www.ncbi.nlm.nih.gov/pubmed/30987646
http://dx.doi.org/10.1186/s12964-019-0345-3

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