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Hypopituitarism After Traumatic Brain Injury
Acquired hypopituitarism is associated with traumatic brain injury. This neuroendocrine dysfunction can cause both short-term and long-term morbidity resulting in a cognitive, physiological, and behavioral decline, which increases the burden of the disease and the cost of care. Data in the trauma li...
Autores principales: | , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Cureus
2019
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6497183/ https://www.ncbi.nlm.nih.gov/pubmed/31065469 http://dx.doi.org/10.7759/cureus.4163 |
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author | Gray, Sanjiv Bilski, Tracy Dieudonne, Beatrice Saeed, Saqib |
author_facet | Gray, Sanjiv Bilski, Tracy Dieudonne, Beatrice Saeed, Saqib |
author_sort | Gray, Sanjiv |
collection | PubMed |
description | Acquired hypopituitarism is associated with traumatic brain injury. This neuroendocrine dysfunction can cause both short-term and long-term morbidity resulting in a cognitive, physiological, and behavioral decline, which increases the burden of the disease and the cost of care. Data in the trauma literature is derisory on this subject. The aim of this review is to edify clinicians on this condition, outline the screening criteria and methods for hypopituitarism after traumatic brain injury, and bring awareness to the chronic effects. |
format | Online Article Text |
id | pubmed-6497183 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2019 |
publisher | Cureus |
record_format | MEDLINE/PubMed |
spelling | pubmed-64971832019-05-07 Hypopituitarism After Traumatic Brain Injury Gray, Sanjiv Bilski, Tracy Dieudonne, Beatrice Saeed, Saqib Cureus Endocrinology/Diabetes/Metabolism Acquired hypopituitarism is associated with traumatic brain injury. This neuroendocrine dysfunction can cause both short-term and long-term morbidity resulting in a cognitive, physiological, and behavioral decline, which increases the burden of the disease and the cost of care. Data in the trauma literature is derisory on this subject. The aim of this review is to edify clinicians on this condition, outline the screening criteria and methods for hypopituitarism after traumatic brain injury, and bring awareness to the chronic effects. Cureus 2019-03-01 /pmc/articles/PMC6497183/ /pubmed/31065469 http://dx.doi.org/10.7759/cureus.4163 Text en Copyright © 2019, Gray et al. http://creativecommons.org/licenses/by/3.0/ This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. |
spellingShingle | Endocrinology/Diabetes/Metabolism Gray, Sanjiv Bilski, Tracy Dieudonne, Beatrice Saeed, Saqib Hypopituitarism After Traumatic Brain Injury |
title | Hypopituitarism After Traumatic Brain Injury |
title_full | Hypopituitarism After Traumatic Brain Injury |
title_fullStr | Hypopituitarism After Traumatic Brain Injury |
title_full_unstemmed | Hypopituitarism After Traumatic Brain Injury |
title_short | Hypopituitarism After Traumatic Brain Injury |
title_sort | hypopituitarism after traumatic brain injury |
topic | Endocrinology/Diabetes/Metabolism |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6497183/ https://www.ncbi.nlm.nih.gov/pubmed/31065469 http://dx.doi.org/10.7759/cureus.4163 |
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