Cargando…
A requirement for septins and the autophagy receptor p62 in the proliferation of intracellular Shigella
Shigella flexneri, a Gram‐negative enteroinvasive pathogen, causes inflammatory destruction of the human intestinal epithelium. During infection of epithelial cells, Shigella escape from the phagosome to the cytosol, where they reroute host cell glycolysis to obtain nutrients for proliferation. Sept...
Autores principales: | Lobato‐Márquez, Damián, Krokowski, Sina, Sirianni, Andrea, Larrouy‐Maumus, Gerald, Mostowy, Serge |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2018
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6519264/ https://www.ncbi.nlm.nih.gov/pubmed/29752866 http://dx.doi.org/10.1002/cm.21453 |
Ejemplares similares
-
Mitochondria mediate septin cage assembly to promote autophagy of Shigella
por: Sirianni, Andrea, et al.
Publicado: (2016) -
Septins Recognize and Entrap Dividing Bacterial Cells for Delivery to Lysosomes
por: Krokowski, Sina, et al.
Publicado: (2018) -
Bacterial cell division is recognized by the septin cytoskeleton for restriction by autophagy
por: Krokowski, Sina, et al.
Publicado: (2019) -
Interactions between Shigella flexneri and the Autophagy Machinery
por: Krokowski, Sina, et al.
Publicado: (2016) -
Intact Cell Lipidomics
Reveal Changes to the Ratio
of Cardiolipins to Phosphatidylinositols in Response to Kanamycin
in HeLa and Primary Cells
por: Rebollo-Ramirez, Sonia, et al.
Publicado: (2018)