Cargando…
Alamandine attenuates long-term hypertension-induced cardiac fibrosis independent of blood pressure
Cardiac fibrosis secondary to long-term hypertension is known to promote cardiac dysfunction; however, few therapeutic agents are available for the treatment of this condition in clinical practice. The heptapeptide alamandine (Ala) has recently been identified as a component of the renin-angiotensin...
Autores principales: | Wang, Lan, Liu, Chi, Chen, Xiru, Li, Peng |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
D.A. Spandidos
2019
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6522836/ https://www.ncbi.nlm.nih.gov/pubmed/31059021 http://dx.doi.org/10.3892/mmr.2019.10167 |
Ejemplares similares
-
Alamandine attenuates hypertension and cardiac hypertrophy in hypertensive rats
por: Liu, Chi, et al.
Publicado: (2018) -
Alamandine alleviates hypertension and renal damage via oxidative-stress attenuation in Dahl rats
por: Gong, Juexiao, et al.
Publicado: (2022) -
Correction: Alamandine attenuates hepatic fibrosis by regulating autophagy induced by NOX4-dependent ROS
Publicado: (2021) -
Alamandine alleviated heart failure and fibrosis in myocardial infarction mice
por: Zhao, Kun, et al.
Publicado: (2022) -
Assessment of Alamandine in Pulmonary Fibrosis and Respiratory Mechanics in Rodents
por: Fernandes, Renata Streck, et al.
Publicado: (2021)