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Cell aging preserves cellular immortality in the presence of lethal levels of damage
Cellular aging, a progressive functional decline driven by damage accumulation, often culminates in the mortality of a cell lineage. Certain lineages, however, are able to sustain long-lasting immortality, as prominently exemplified by stem cells. Here, we show that Escherichia coli cell lineages ex...
Autores principales: | , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2019
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6532838/ https://www.ncbi.nlm.nih.gov/pubmed/31120870 http://dx.doi.org/10.1371/journal.pbio.3000266 |
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author | Proenca, Audrey Menegaz Rang, Camilla Ulla Qiu, Andrew Shi, Chao Chao, Lin |
author_facet | Proenca, Audrey Menegaz Rang, Camilla Ulla Qiu, Andrew Shi, Chao Chao, Lin |
author_sort | Proenca, Audrey Menegaz |
collection | PubMed |
description | Cellular aging, a progressive functional decline driven by damage accumulation, often culminates in the mortality of a cell lineage. Certain lineages, however, are able to sustain long-lasting immortality, as prominently exemplified by stem cells. Here, we show that Escherichia coli cell lineages exhibit comparable patterns of mortality and immortality. Through single-cell microscopy and microfluidic techniques, we find that these patterns are explained by the dynamics of damage accumulation and asymmetric partitioning between daughter cells. At low damage accumulation rates, both aging and rejuvenating lineages retain immortality by reaching their respective states of physiological equilibrium. We show that both asymmetry and equilibrium are present in repair mutants lacking certain repair chaperones, suggesting that intact repair capacity is not essential for immortal proliferation. We show that this growth equilibrium, however, is displaced by extrinsic damage in a dosage-dependent response. Moreover, we demonstrate that aging lineages become mortal when damage accumulation rates surpass a threshold, whereas rejuvenating lineages within the same population remain immortal. Thus, the processes of damage accumulation and partitioning through asymmetric cell division are essential in the determination of proliferative mortality and immortality in bacterial populations. This study provides further evidence for the characterization of cellular aging as a general process, affecting prokaryotes and eukaryotes alike and according to similar evolutionary constraints. |
format | Online Article Text |
id | pubmed-6532838 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2019 |
publisher | Public Library of Science |
record_format | MEDLINE/PubMed |
spelling | pubmed-65328382019-06-05 Cell aging preserves cellular immortality in the presence of lethal levels of damage Proenca, Audrey Menegaz Rang, Camilla Ulla Qiu, Andrew Shi, Chao Chao, Lin PLoS Biol Research Article Cellular aging, a progressive functional decline driven by damage accumulation, often culminates in the mortality of a cell lineage. Certain lineages, however, are able to sustain long-lasting immortality, as prominently exemplified by stem cells. Here, we show that Escherichia coli cell lineages exhibit comparable patterns of mortality and immortality. Through single-cell microscopy and microfluidic techniques, we find that these patterns are explained by the dynamics of damage accumulation and asymmetric partitioning between daughter cells. At low damage accumulation rates, both aging and rejuvenating lineages retain immortality by reaching their respective states of physiological equilibrium. We show that both asymmetry and equilibrium are present in repair mutants lacking certain repair chaperones, suggesting that intact repair capacity is not essential for immortal proliferation. We show that this growth equilibrium, however, is displaced by extrinsic damage in a dosage-dependent response. Moreover, we demonstrate that aging lineages become mortal when damage accumulation rates surpass a threshold, whereas rejuvenating lineages within the same population remain immortal. Thus, the processes of damage accumulation and partitioning through asymmetric cell division are essential in the determination of proliferative mortality and immortality in bacterial populations. This study provides further evidence for the characterization of cellular aging as a general process, affecting prokaryotes and eukaryotes alike and according to similar evolutionary constraints. Public Library of Science 2019-05-23 /pmc/articles/PMC6532838/ /pubmed/31120870 http://dx.doi.org/10.1371/journal.pbio.3000266 Text en © 2019 Proenca et al http://creativecommons.org/licenses/by/4.0/ This is an open access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/) , which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. |
spellingShingle | Research Article Proenca, Audrey Menegaz Rang, Camilla Ulla Qiu, Andrew Shi, Chao Chao, Lin Cell aging preserves cellular immortality in the presence of lethal levels of damage |
title | Cell aging preserves cellular immortality in the presence of lethal levels of damage |
title_full | Cell aging preserves cellular immortality in the presence of lethal levels of damage |
title_fullStr | Cell aging preserves cellular immortality in the presence of lethal levels of damage |
title_full_unstemmed | Cell aging preserves cellular immortality in the presence of lethal levels of damage |
title_short | Cell aging preserves cellular immortality in the presence of lethal levels of damage |
title_sort | cell aging preserves cellular immortality in the presence of lethal levels of damage |
topic | Research Article |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6532838/ https://www.ncbi.nlm.nih.gov/pubmed/31120870 http://dx.doi.org/10.1371/journal.pbio.3000266 |
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