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Myocardial death and dysfunction after ischemia-reperfusion injury require CaMKIIδ oxidation
Reactive oxygen species (ROS) contribute to myocardial death during ischemia-reperfusion (I/R) injury, but detailed knowledge of molecular pathways connecting ROS to cardiac injury is lacking. Activation of the Ca(2+)/calmodulin-dependent protein kinase II (CaMKIIδ) is implicated in myocardial death...
| Autores principales: | , , , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
| Publicado: |
Nature Publishing Group UK
2019
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6595001/ https://www.ncbi.nlm.nih.gov/pubmed/31243295 http://dx.doi.org/10.1038/s41598-019-45743-6 |