Organophosphorus-Induced Toxic Myeloneuropathy: Series of Three Adolescent Patients with Short Review

Organophosphate (OP) poisoning is the most common poisoning in India, accounting for almost half of the hospital admissions due to poisoning. Delayed neuropathy is initiated by an attack on a nervous tissue esterase. Although uncommon, delayed neurotoxicity has been consistently reported in literatu...

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Autores principales: Nayak, Priyabrata, Mallick, Ashok K, Mishra, Shubhankar, Panigrahy, Debasish
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Wolters Kluwer - Medknow 2019
Materias:
Case Report
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6601111/
https://www.ncbi.nlm.nih.gov/pubmed/31316643
http://dx.doi.org/10.4103/jpn.JPN_45_18
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author Nayak, Priyabrata
Mallick, Ashok K
Mishra, Shubhankar
Panigrahy, Debasish
author_facet Nayak, Priyabrata
Mallick, Ashok K
Mishra, Shubhankar
Panigrahy, Debasish
author_sort Nayak, Priyabrata
collection PubMed
description Organophosphate (OP) poisoning is the most common poisoning in India, accounting for almost half of the hospital admissions due to poisoning. Delayed neuropathy is initiated by an attack on a nervous tissue esterase. Although uncommon, delayed neurotoxicity has been consistently reported in literature. This mechanism is implicated not only in damaging peripheral nervous system but also in causing central processes leading to myelopathy. We report a series of three adolescent patients who came to our hospital with delayed neurological manifestations of organophosphorus poisoning, which came out to be OP-induced myeloneuropathy after detailed analysis and evaluation.
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spelling pubmed-66011112019-07-17 Organophosphorus-Induced Toxic Myeloneuropathy: Series of Three Adolescent Patients with Short Review Nayak, Priyabrata Mallick, Ashok K Mishra, Shubhankar Panigrahy, Debasish J Pediatr Neurosci Case Report Organophosphate (OP) poisoning is the most common poisoning in India, accounting for almost half of the hospital admissions due to poisoning. Delayed neuropathy is initiated by an attack on a nervous tissue esterase. Although uncommon, delayed neurotoxicity has been consistently reported in literature. This mechanism is implicated not only in damaging peripheral nervous system but also in causing central processes leading to myelopathy. We report a series of three adolescent patients who came to our hospital with delayed neurological manifestations of organophosphorus poisoning, which came out to be OP-induced myeloneuropathy after detailed analysis and evaluation. Wolters Kluwer - Medknow 2019 /pmc/articles/PMC6601111/ /pubmed/31316643 http://dx.doi.org/10.4103/jpn.JPN_45_18 Text en Copyright: © 2019 Journal of Pediatric Neurosciences http://creativecommons.org/licenses/by-nc-sa/4.0 This is an open access journal, and articles are distributed under the terms of the Creative Commons Attribution-NonCommercial-ShareAlike 4.0 License, which allows others to remix, tweak, and build upon the work non-commercially, as long as appropriate credit is given and the new creations are licensed under the identical terms.
spellingShingle Case Report
Nayak, Priyabrata
Mallick, Ashok K
Mishra, Shubhankar
Panigrahy, Debasish
Organophosphorus-Induced Toxic Myeloneuropathy: Series of Three Adolescent Patients with Short Review
title Organophosphorus-Induced Toxic Myeloneuropathy: Series of Three Adolescent Patients with Short Review
title_full Organophosphorus-Induced Toxic Myeloneuropathy: Series of Three Adolescent Patients with Short Review
title_fullStr Organophosphorus-Induced Toxic Myeloneuropathy: Series of Three Adolescent Patients with Short Review
title_full_unstemmed Organophosphorus-Induced Toxic Myeloneuropathy: Series of Three Adolescent Patients with Short Review
title_short Organophosphorus-Induced Toxic Myeloneuropathy: Series of Three Adolescent Patients with Short Review
title_sort organophosphorus-induced toxic myeloneuropathy: series of three adolescent patients with short review
topic Case Report
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6601111/
https://www.ncbi.nlm.nih.gov/pubmed/31316643
http://dx.doi.org/10.4103/jpn.JPN_45_18
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