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Homocysteinemia as a cause for amaurosis fugax in a patient without an apparent embolic source

Purpose: To report a case with monocular transient vision loss (TVL) associated with Hyperhomocysteinemia. Methods: We present a case with persistent TVL attacks and high level of homocysteine. Results: A 32-year-old male had a history of episodes of recurrent monocular TVL. Extensive ophthalmic, sy...

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Autores principales: Inan, Sibel, Yavas, Guliz, Inan, Umit Ubeyt
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Romanian Society of Ophthalmology 2019
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6626925/
https://www.ncbi.nlm.nih.gov/pubmed/31334400
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author Inan, Sibel
Yavas, Guliz
Inan, Umit Ubeyt
author_facet Inan, Sibel
Yavas, Guliz
Inan, Umit Ubeyt
author_sort Inan, Sibel
collection PubMed
description Purpose: To report a case with monocular transient vision loss (TVL) associated with Hyperhomocysteinemia. Methods: We present a case with persistent TVL attacks and high level of homocysteine. Results: A 32-year-old male had a history of episodes of recurrent monocular TVL. Extensive ophthalmic, systemic and laboratory studies were unremarkable with the exception of high plasma homocysteine level. He never experienced TVL during the 36-month follow-up after starting folate, B12 and B6 except for one episode in which he had discontinued the treatment for three months. Conclusion: This case may suggest hyperhomocysteinemia as one of the underlying causes of recurrent attacks of TVL without any known source of emboli.
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spelling pubmed-66269252019-07-22 Homocysteinemia as a cause for amaurosis fugax in a patient without an apparent embolic source Inan, Sibel Yavas, Guliz Inan, Umit Ubeyt Rom J Ophthalmol Case Reports Purpose: To report a case with monocular transient vision loss (TVL) associated with Hyperhomocysteinemia. Methods: We present a case with persistent TVL attacks and high level of homocysteine. Results: A 32-year-old male had a history of episodes of recurrent monocular TVL. Extensive ophthalmic, systemic and laboratory studies were unremarkable with the exception of high plasma homocysteine level. He never experienced TVL during the 36-month follow-up after starting folate, B12 and B6 except for one episode in which he had discontinued the treatment for three months. Conclusion: This case may suggest hyperhomocysteinemia as one of the underlying causes of recurrent attacks of TVL without any known source of emboli. Romanian Society of Ophthalmology 2019 /pmc/articles/PMC6626925/ /pubmed/31334400 Text en ©Romanian Society of Ophthalmology http://creativecommons.org/licenses/by/2.0/ This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Case Reports
Inan, Sibel
Yavas, Guliz
Inan, Umit Ubeyt
Homocysteinemia as a cause for amaurosis fugax in a patient without an apparent embolic source
title Homocysteinemia as a cause for amaurosis fugax in a patient without an apparent embolic source
title_full Homocysteinemia as a cause for amaurosis fugax in a patient without an apparent embolic source
title_fullStr Homocysteinemia as a cause for amaurosis fugax in a patient without an apparent embolic source
title_full_unstemmed Homocysteinemia as a cause for amaurosis fugax in a patient without an apparent embolic source
title_short Homocysteinemia as a cause for amaurosis fugax in a patient without an apparent embolic source
title_sort homocysteinemia as a cause for amaurosis fugax in a patient without an apparent embolic source
topic Case Reports
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6626925/
https://www.ncbi.nlm.nih.gov/pubmed/31334400
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