Cargando…
Store-Operated Ca(2+) Entry in Tumor Progression: From Molecular Mechanisms to Clinical Implications
The remodeling of Ca(2+) homeostasis has been implicated as a critical event in driving malignant phenotypes, such as tumor cell proliferation, motility, and metastasis. Store-operated Ca(2+) entry (SOCE) that is elicited by the depletion of the endoplasmic reticulum (ER) Ca(2+) stores constitutes t...
Autores principales: | Chen, Yih-Fung, Lin, Peng-Chan, Yeh, Yu-Min, Chen, Li-Hsien, Shen, Meng-Ru |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2019
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6678533/ https://www.ncbi.nlm.nih.gov/pubmed/31252656 http://dx.doi.org/10.3390/cancers11070899 |
Ejemplares similares
-
The STIM1-Orai1 pathway of store-operated Ca(2+) entry controls the checkpoint in cell cycle G1/S transition
por: Chen, Yun-Wen, et al.
Publicado: (2016) -
Regulation of Store-Operated Ca(2+) Entry by Septins
por: Deb, Bipan K., et al.
Publicado: (2016) -
Regulation of Store-Operated Ca(2+) Entry by SARAF
por: Dagan, Inbal, et al.
Publicado: (2021) -
Gastrodin Inhibits Store-Operated Ca(2+) Entry and Alleviates Cardiac Hypertrophy
por: Zheng, Changbo, et al.
Publicado: (2017) -
Store-operated Ca(2+) entry in muscle physiology and diseases
por: Pan, Zui, et al.
Publicado: (2014)