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Cilostazol inhibits hyperglucose-induced vascular smooth muscle cell dysfunction by modulating the RAGE/ERK/NF-κB signaling pathways
BACKGROUND: Increasing evidence suggests that high glucose (HG) causes abnormalities in endothelial and vascular smooth muscle cell function (VSMC) and contributes to atherosclerosis. Receptor for advanced glycation end-products (RAGE) has been linked to the pathogenesis of both the macrovascular an...
Autores principales: | Su, Sheng-Chiang, Hung, Yi-Jen, Huang, Chia-Luen, Shieh, Yi-Shing, Chien, Chu-Yen, Chiang, Chi-Fu, Liu, Jhih-Syuan, Lu, Chieh-Hua, Hsieh, Chang-Hsun, Lin, Chien-Ming, Lee, Chien-Hsing |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2019
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6731603/ https://www.ncbi.nlm.nih.gov/pubmed/31492153 http://dx.doi.org/10.1186/s12929-019-0550-9 |
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