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Diabetes causal attributions among affected and unaffected individuals
OBJECTIVE: The present study aims to describe and compare causal attributions for type 1 diabetes (T1D) and type 2 diabetes (T2D) among affected and unaffected individuals and to investigate the relationships among attributions, attitudes, and beliefs. RESEARCH DESIGN AND METHODS: Adults with no dia...
Autores principales: | , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BMJ Publishing Group
2019
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6731786/ https://www.ncbi.nlm.nih.gov/pubmed/31543977 http://dx.doi.org/10.1136/bmjdrc-2019-000708 |
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author | Rose, Margaret K Costabile, Kristi A Boland, Sarah E Cohen, Rachel W Persky, Susan |
author_facet | Rose, Margaret K Costabile, Kristi A Boland, Sarah E Cohen, Rachel W Persky, Susan |
author_sort | Rose, Margaret K |
collection | PubMed |
description | OBJECTIVE: The present study aims to describe and compare causal attributions for type 1 diabetes (T1D) and type 2 diabetes (T2D) among affected and unaffected individuals and to investigate the relationships among attributions, attitudes, and beliefs. RESEARCH DESIGN AND METHODS: Adults with no diabetes (N=458), T1D (N=192), or T2D (N=207) completed an online survey. Measures assessed diabetes conceptual knowledge, causal attributions for T1D and T2D, perceived control over diabetes onset, and favorability judgements of individuals affected by each type. RESULTS: Results indicate general agreement on causal attributions for T1D and T2D among all respondent groups, with some divergences by disease status. All respondents attributed both T1D and T2D to genetics, and genetic attributions were positively associated with favorability judgements of individuals with T2D, but not those with T1D. CONCLUSIONS: This report sets the stage for investigations into how and why attributions for T1D and T2D differ and the implications of these differences including stigmatization of individuals with diabetes and diabetes-related self-concept. Additionally, this work can inform efforts towards clinical and public health education to prevent and optimize treatment of T1D and T2D. |
format | Online Article Text |
id | pubmed-6731786 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2019 |
publisher | BMJ Publishing Group |
record_format | MEDLINE/PubMed |
spelling | pubmed-67317862019-09-20 Diabetes causal attributions among affected and unaffected individuals Rose, Margaret K Costabile, Kristi A Boland, Sarah E Cohen, Rachel W Persky, Susan BMJ Open Diabetes Res Care Psychosocial Research OBJECTIVE: The present study aims to describe and compare causal attributions for type 1 diabetes (T1D) and type 2 diabetes (T2D) among affected and unaffected individuals and to investigate the relationships among attributions, attitudes, and beliefs. RESEARCH DESIGN AND METHODS: Adults with no diabetes (N=458), T1D (N=192), or T2D (N=207) completed an online survey. Measures assessed diabetes conceptual knowledge, causal attributions for T1D and T2D, perceived control over diabetes onset, and favorability judgements of individuals affected by each type. RESULTS: Results indicate general agreement on causal attributions for T1D and T2D among all respondent groups, with some divergences by disease status. All respondents attributed both T1D and T2D to genetics, and genetic attributions were positively associated with favorability judgements of individuals with T2D, but not those with T1D. CONCLUSIONS: This report sets the stage for investigations into how and why attributions for T1D and T2D differ and the implications of these differences including stigmatization of individuals with diabetes and diabetes-related self-concept. Additionally, this work can inform efforts towards clinical and public health education to prevent and optimize treatment of T1D and T2D. BMJ Publishing Group 2019-08-30 /pmc/articles/PMC6731786/ /pubmed/31543977 http://dx.doi.org/10.1136/bmjdrc-2019-000708 Text en © Author(s) (or their employer(s)) 2019. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/. |
spellingShingle | Psychosocial Research Rose, Margaret K Costabile, Kristi A Boland, Sarah E Cohen, Rachel W Persky, Susan Diabetes causal attributions among affected and unaffected individuals |
title | Diabetes causal attributions among affected and unaffected individuals |
title_full | Diabetes causal attributions among affected and unaffected individuals |
title_fullStr | Diabetes causal attributions among affected and unaffected individuals |
title_full_unstemmed | Diabetes causal attributions among affected and unaffected individuals |
title_short | Diabetes causal attributions among affected and unaffected individuals |
title_sort | diabetes causal attributions among affected and unaffected individuals |
topic | Psychosocial Research |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6731786/ https://www.ncbi.nlm.nih.gov/pubmed/31543977 http://dx.doi.org/10.1136/bmjdrc-2019-000708 |
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