Cargando…
Macrophages facilitate post myocardial infarction arrhythmias: roles of gap junction and KCa3.1
Effective therapeutic targets against post-myocardial infarction (MI) arrhythmias remain to be discovered. We aimed to investigate the role of macrophages in post-MI arrhythmias. Methods: Mononuclear cell accumulation, macrophage polarization from M0 to M1 subset, and gap junction formation were ana...
Autores principales: | Fei, Yu-Dong, Wang, Qian, Hou, Jian-Wen, Li, Wei, Cai, Xing-Xing, Yang, Yu-Li, Zhang, Liu-Hui, Wei, Zhi-Xing, Chen, Tai-Zhong, Wang, Yue-Peng, Li, Yi-Gang |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Ivyspring International Publisher
2019
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6771231/ https://www.ncbi.nlm.nih.gov/pubmed/31588225 http://dx.doi.org/10.7150/thno.34801 |
Ejemplares similares
-
Contribution of the KCa3.1 channel–calmodulin interactions to the regulation of the KCa3.1 gating process
por: Morales, Patricia, et al.
Publicado: (2013) -
KCa3.1 inhibition switches the phenotype of glioma-infiltrating microglia/macrophages
por: Grimaldi, A, et al.
Publicado: (2016) -
Electrocardiogram Heartbeat Classification for Arrhythmias and Myocardial Infarction
por: Pham, Bach-Tung, et al.
Publicado: (2023) -
Enhancement of Gap Junction Function During Acute Myocardial Infarction Modifies Healing and Reduces Late Ventricular Arrhythmia Susceptibility
por: Ng, Fu Siong, et al.
Publicado: (2016) -
KCa3.1 Inhibition of Macrophages Suppresses Inflammatory Response Leading to Endothelial Damage in a Cell Model of Kawasaki Disease
por: Zheng, Fenglei, et al.
Publicado: (2021)