N-Glycosylation of the Discoidin Domain Receptor Is Required for Axon Regeneration in Caenorhabditis elegans

Axon regeneration following neuronal injury is an important repair mechanism that is not well understood at present. In Caenorhabditis elegans, axon regeneration is regulated by DDR-2, a receptor tyrosine kinase (RTK) that contains a discoidin domain and modulates the Met-like SVH-2 RTK–JNK MAP kina...

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Detalles Bibliográficos
Autores principales: Shimizu, Tatsuhiro, Kato, Yuka, Sakai, Yoshiki, Hisamoto, Naoki, Matsumoto, Kunihiro
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Genetics Society of America 2019
Materias:
Investigations
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6781908/
https://www.ncbi.nlm.nih.gov/pubmed/31371405
http://dx.doi.org/10.1534/genetics.119.302492

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6781908/
https://www.ncbi.nlm.nih.gov/pubmed/31371405
http://dx.doi.org/10.1534/genetics.119.302492

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