DNA mismatch repair controls the host innate response and cell fate after influenza virus infection

Despite the cytopathic nature of influenza A virus (IAV) replication, we recently reported that a subset of lung epithelial club cells is able to intrinsically clear virus and survive infection. However, the mechanisms that drive cell survival during a normally lytic infection remained unclear. Usin...

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Detalles Bibliográficos
Autores principales: Chambers, Benjamin S., Heaton, Brook E., Rausch, Keiko, Dumm, Rebekah E., Hamilton, Jennifer R., Cherry, Sara, Heaton, Nicholas S.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: 2019
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6814535/
https://www.ncbi.nlm.nih.gov/pubmed/31358986
http://dx.doi.org/10.1038/s41564-019-0509-3

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6814535/
https://www.ncbi.nlm.nih.gov/pubmed/31358986
http://dx.doi.org/10.1038/s41564-019-0509-3

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