Cargando…
Exosomes from nicotine-stimulated macrophages accelerate atherosclerosis through miR-21-3p/PTEN-mediated VSMC migration and proliferation
Rationale: During the development of atherosclerosis, macrophages secrete exosomes that regulate vascular smooth muscle cells (VSMCs); however, whether nicotine, a major constituent of cigarettes, can modulate this communication in the context of atherogenesis remains to be further studied. In this...
Autores principales: | Zhu, Jumo, Liu, Bei, Wang, Zhiyan, Wang, Di, Ni, Huaner, Zhang, Lili, Wang, Yi |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Ivyspring International Publisher
2019
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6815950/ https://www.ncbi.nlm.nih.gov/pubmed/31660076 http://dx.doi.org/10.7150/thno.37357 |
Ejemplares similares
-
The interaction between STAT3 and nAChRα1 interferes with nicotine-induced atherosclerosis via Akt/mTOR signaling cascade
por: Xu, Shuang, et al.
Publicado: (2019) -
Nicotine facilitates VSMC dysfunction through a miR-200b/RhoGDIA/cytoskeleton module
por: Liang, Dongli, et al.
Publicado: (2017) -
Exosomes in atherosclerosis: Convergence on macrophages
por: Yang, Kaiying, et al.
Publicado: (2022) -
Platelet-derived microvesicles induce calcium oscillations and promote VSMC migration via TRPV4
por: Li, Shan-Shan, et al.
Publicado: (2021) -
Pitavastatin Regulates Ang II Induced Proliferation and Migration via IGFBP-5 in VSMC
por: Ha, Yu Mi, et al.
Publicado: (2015)