BTXA regulates the epithelial–mesenchymal transition and autophagy of keloid fibroblasts via modulating miR-1587/miR-2392 targeted ZEB2

Keloids are very resistant to treatment in dermatology and plastic surgical practice. The present study aimed to explore the underlying mechanism of botulinum toxin A (BTXA) treated human skin keloid fibroblasts (HSFBs) proving some new insights into keloids treatment. Expression of miR-1587 and miR...

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Detalles Bibliográficos
Autores principales: Hou, Zhanying, Fan, Feixiang, Liu, Po
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Portland Press Ltd. 2019
Materias:
Cell Death & Injury
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6822502/
https://www.ncbi.nlm.nih.gov/pubmed/31652445
http://dx.doi.org/10.1042/BSR20190679

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6822502/
https://www.ncbi.nlm.nih.gov/pubmed/31652445
http://dx.doi.org/10.1042/BSR20190679

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