Cargando…
miR- 26a Sensitizes Melanoma Cells To Dabrafenib Via Targeting HMGB1-Dependent Autophagy Pathways
BACKGROUND: Melanoma is known as the most aggressive and lethal type of cutaneous cancer due to its rapid development of drug resistance to chemotherapy drugs. METHODS: In our study, we conducted a variety of studies, including quantitative PCR, Western blot, and autophagy and apoptosis assays to in...
Autores principales: | Yu, Yan, Xiang, Niu, Lin, Min, Huang, Jin-Wen, Zhang, Jing, Cheng, Bo, Ji, Chao |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Dove
2019
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6825511/ https://www.ncbi.nlm.nih.gov/pubmed/31754297 http://dx.doi.org/10.2147/DDDT.S225671 |
Ejemplares similares
-
Endoplasmic reticulum stress-induced autophagy determines the susceptibility of melanoma cells to dabrafenib
por: Ji, Chao, et al.
Publicado: (2016) -
Identification of Key miRNAs in the Treatment of Dabrafenib-Resistant Melanoma
por: Gao, Guangyu, et al.
Publicado: (2021) -
Anti-septic effects of dabrafenib on HMGB1-mediated inflammatory responses
por: Jung, Byeongjin, et al.
Publicado: (2016) -
Dabrafenib for treatment of BRAF-mutant melanoma
por: Kainthla, Radhika, et al.
Publicado: (2013) -
Endoplasmic Reticulum Stress-Induced Autophagy Determines the Susceptibility of Melanoma Cells to Dabrafenib [Retraction]
Publicado: (2023)