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Replication Stress Response Links RAD52 to Protecting Common Fragile Sites
Rad52 in yeast is a key player in homologous recombination (HR), but mammalian RAD52 is dispensable for HR as shown by the lack of a strong HR phenotype in RAD52-deficient cells and in RAD52 knockout mice. RAD52 function in mammalian cells first emerged with the discovery of its important backup rol...
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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MDPI
2019
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6826974/ https://www.ncbi.nlm.nih.gov/pubmed/31569559 http://dx.doi.org/10.3390/cancers11101467 |