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Anterograde Amnesia after Acute Glufosinate Ammonium Intoxication

Glufosinate ammonium (GA) intoxication causes several neurologic complications. We report a rare but typical case of GA intoxication associated with anterograde amnesia and bilateral hippocampal involvement. A 53-year-old woman with GA intoxication presented to the emergency department. Initial gene...

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Autores principales: Kim, Hyuk-Hoon, Min, Young-Gi
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Korean Society of Critical Care Medicine 2018
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6849054/
https://www.ncbi.nlm.nih.gov/pubmed/31723872
http://dx.doi.org/10.4266/acc.2016.00444
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author Kim, Hyuk-Hoon
Min, Young-Gi
author_facet Kim, Hyuk-Hoon
Min, Young-Gi
author_sort Kim, Hyuk-Hoon
collection PubMed
description Glufosinate ammonium (GA) intoxication causes several neurologic complications. We report a rare but typical case of GA intoxication associated with anterograde amnesia and bilateral hippocampal involvement. A 53-year-old woman with GA intoxication presented to the emergency department. Initial general and neurologic examinations were unremarkable but, from the day after admission, she exhibited anterograde amnesia. On brain magnetic resonance imaging, the signal intensity in the hippocampus was symmetrically and bilaterally increased. She was discharged with no medical problems, but the anterograde amnesia remained. Eleven days after the onset of amnesia, she returned to the neurology outpatient department with persisting anterograde amnesia but improving symptoms.
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spelling pubmed-68490542019-11-13 Anterograde Amnesia after Acute Glufosinate Ammonium Intoxication Kim, Hyuk-Hoon Min, Young-Gi Acute Crit Care Case Report Glufosinate ammonium (GA) intoxication causes several neurologic complications. We report a rare but typical case of GA intoxication associated with anterograde amnesia and bilateral hippocampal involvement. A 53-year-old woman with GA intoxication presented to the emergency department. Initial general and neurologic examinations were unremarkable but, from the day after admission, she exhibited anterograde amnesia. On brain magnetic resonance imaging, the signal intensity in the hippocampus was symmetrically and bilaterally increased. She was discharged with no medical problems, but the anterograde amnesia remained. Eleven days after the onset of amnesia, she returned to the neurology outpatient department with persisting anterograde amnesia but improving symptoms. Korean Society of Critical Care Medicine 2018-05 2018-05-31 /pmc/articles/PMC6849054/ /pubmed/31723872 http://dx.doi.org/10.4266/acc.2016.00444 Text en Copyright © 2018 The Korean Society of Critical Care Medicine This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Case Report
Kim, Hyuk-Hoon
Min, Young-Gi
Anterograde Amnesia after Acute Glufosinate Ammonium Intoxication
title Anterograde Amnesia after Acute Glufosinate Ammonium Intoxication
title_full Anterograde Amnesia after Acute Glufosinate Ammonium Intoxication
title_fullStr Anterograde Amnesia after Acute Glufosinate Ammonium Intoxication
title_full_unstemmed Anterograde Amnesia after Acute Glufosinate Ammonium Intoxication
title_short Anterograde Amnesia after Acute Glufosinate Ammonium Intoxication
title_sort anterograde amnesia after acute glufosinate ammonium intoxication
topic Case Report
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6849054/
https://www.ncbi.nlm.nih.gov/pubmed/31723872
http://dx.doi.org/10.4266/acc.2016.00444
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