Cargando…
Retraction Note to: Excessive mitochondrial fragmentation triggered by erlotinib promotes pancreatic cancer PANC‑1 cell apoptosis via activating the mROS‑HtrA2/Omi pathways
Autores principales: | , , , , , , , |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2019
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6849270/ https://www.ncbi.nlm.nih.gov/pubmed/31736657 http://dx.doi.org/10.1186/s12935-019-1019-3 |
_version_ | 1783469175031726080 |
---|---|
author | Wan, Jun Cui, Jie Wang, Lei Wu, Kunpeng Hong, Xiaoping Zou, Yulin Zhao, Shuang Ke, Hong |
author_facet | Wan, Jun Cui, Jie Wang, Lei Wu, Kunpeng Hong, Xiaoping Zou, Yulin Zhao, Shuang Ke, Hong |
author_sort | Wan, Jun |
collection | PubMed |
description | |
format | Online Article Text |
id | pubmed-6849270 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2019 |
publisher | BioMed Central |
record_format | MEDLINE/PubMed |
spelling | pubmed-68492702019-11-15 Retraction Note to: Excessive mitochondrial fragmentation triggered by erlotinib promotes pancreatic cancer PANC‑1 cell apoptosis via activating the mROS‑HtrA2/Omi pathways Wan, Jun Cui, Jie Wang, Lei Wu, Kunpeng Hong, Xiaoping Zou, Yulin Zhao, Shuang Ke, Hong Cancer Cell Int Retraction Note BioMed Central 2019-11-12 /pmc/articles/PMC6849270/ /pubmed/31736657 http://dx.doi.org/10.1186/s12935-019-1019-3 Text en © The Author(s) 2019 Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated. |
spellingShingle | Retraction Note Wan, Jun Cui, Jie Wang, Lei Wu, Kunpeng Hong, Xiaoping Zou, Yulin Zhao, Shuang Ke, Hong Retraction Note to: Excessive mitochondrial fragmentation triggered by erlotinib promotes pancreatic cancer PANC‑1 cell apoptosis via activating the mROS‑HtrA2/Omi pathways |
title | Retraction Note to: Excessive mitochondrial fragmentation triggered by erlotinib promotes pancreatic cancer PANC‑1 cell apoptosis via activating the mROS‑HtrA2/Omi pathways |
title_full | Retraction Note to: Excessive mitochondrial fragmentation triggered by erlotinib promotes pancreatic cancer PANC‑1 cell apoptosis via activating the mROS‑HtrA2/Omi pathways |
title_fullStr | Retraction Note to: Excessive mitochondrial fragmentation triggered by erlotinib promotes pancreatic cancer PANC‑1 cell apoptosis via activating the mROS‑HtrA2/Omi pathways |
title_full_unstemmed | Retraction Note to: Excessive mitochondrial fragmentation triggered by erlotinib promotes pancreatic cancer PANC‑1 cell apoptosis via activating the mROS‑HtrA2/Omi pathways |
title_short | Retraction Note to: Excessive mitochondrial fragmentation triggered by erlotinib promotes pancreatic cancer PANC‑1 cell apoptosis via activating the mROS‑HtrA2/Omi pathways |
title_sort | retraction note to: excessive mitochondrial fragmentation triggered by erlotinib promotes pancreatic cancer panc‑1 cell apoptosis via activating the mros‑htra2/omi pathways |
topic | Retraction Note |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6849270/ https://www.ncbi.nlm.nih.gov/pubmed/31736657 http://dx.doi.org/10.1186/s12935-019-1019-3 |
work_keys_str_mv | AT wanjun retractionnotetoexcessivemitochondrialfragmentationtriggeredbyerlotinibpromotespancreaticcancerpanc1cellapoptosisviaactivatingthemroshtra2omipathways AT cuijie retractionnotetoexcessivemitochondrialfragmentationtriggeredbyerlotinibpromotespancreaticcancerpanc1cellapoptosisviaactivatingthemroshtra2omipathways AT wanglei retractionnotetoexcessivemitochondrialfragmentationtriggeredbyerlotinibpromotespancreaticcancerpanc1cellapoptosisviaactivatingthemroshtra2omipathways AT wukunpeng retractionnotetoexcessivemitochondrialfragmentationtriggeredbyerlotinibpromotespancreaticcancerpanc1cellapoptosisviaactivatingthemroshtra2omipathways AT hongxiaoping retractionnotetoexcessivemitochondrialfragmentationtriggeredbyerlotinibpromotespancreaticcancerpanc1cellapoptosisviaactivatingthemroshtra2omipathways AT zouyulin retractionnotetoexcessivemitochondrialfragmentationtriggeredbyerlotinibpromotespancreaticcancerpanc1cellapoptosisviaactivatingthemroshtra2omipathways AT zhaoshuang retractionnotetoexcessivemitochondrialfragmentationtriggeredbyerlotinibpromotespancreaticcancerpanc1cellapoptosisviaactivatingthemroshtra2omipathways AT kehong retractionnotetoexcessivemitochondrialfragmentationtriggeredbyerlotinibpromotespancreaticcancerpanc1cellapoptosisviaactivatingthemroshtra2omipathways |