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Management in Fallot Tetralogy associated with Congenital Scoliosis Case Report

Tetralogy of Fallot is the most common congenital heart malformation that produces cyanosis. It consists of four different defects of the heart: ventricular septal defect, pulmonary artery stenosis (blockage of blood flow from the right ventricle to the lungs), right ventricle hypertrophy and dextro...

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Autor principal: Ximena, Stanev
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Carol Davila University Press 2012
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6880200/
https://www.ncbi.nlm.nih.gov/pubmed/31803303
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author Ximena, Stanev
author_facet Ximena, Stanev
author_sort Ximena, Stanev
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description Tetralogy of Fallot is the most common congenital heart malformation that produces cyanosis. It consists of four different defects of the heart: ventricular septal defect, pulmonary artery stenosis (blockage of blood flow from the right ventricle to the lungs), right ventricle hypertrophy and dextroposition of aorta. Echocardiography is essential in establishing the diagnosis of patients with cardiac malformation. Patients with Fallot tetralogy present a higher frequency of major non-cardiac congenital disorders. The association with congenital scoliosis influences vital and functional overcomes, restricting physical activity and lowering life expectancy. The author presents therapeutic management on the clinical case of a 13-year-old child with Fallot tetralogy and congenital scoliosis. Therapeutic management of both illnesses consisted in serial surgical interventions as it follows: first time at the age of six years, cardiac malformation was solved, and later, at the age of 13 years the correction of spinal deformity was performed.
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spelling pubmed-68802002019-12-04 Management in Fallot Tetralogy associated with Congenital Scoliosis Case Report Ximena, Stanev J Med Life Original Article Tetralogy of Fallot is the most common congenital heart malformation that produces cyanosis. It consists of four different defects of the heart: ventricular septal defect, pulmonary artery stenosis (blockage of blood flow from the right ventricle to the lungs), right ventricle hypertrophy and dextroposition of aorta. Echocardiography is essential in establishing the diagnosis of patients with cardiac malformation. Patients with Fallot tetralogy present a higher frequency of major non-cardiac congenital disorders. The association with congenital scoliosis influences vital and functional overcomes, restricting physical activity and lowering life expectancy. The author presents therapeutic management on the clinical case of a 13-year-old child with Fallot tetralogy and congenital scoliosis. Therapeutic management of both illnesses consisted in serial surgical interventions as it follows: first time at the age of six years, cardiac malformation was solved, and later, at the age of 13 years the correction of spinal deformity was performed. Carol Davila University Press 2012 /pmc/articles/PMC6880200/ /pubmed/31803303 Text en ©Carol Davila University Press This article is distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/3.0/), which permits unrestricted use and redistribution provided that the original author and source are credited.
spellingShingle Original Article
Ximena, Stanev
Management in Fallot Tetralogy associated with Congenital Scoliosis Case Report
title Management in Fallot Tetralogy associated with Congenital Scoliosis Case Report
title_full Management in Fallot Tetralogy associated with Congenital Scoliosis Case Report
title_fullStr Management in Fallot Tetralogy associated with Congenital Scoliosis Case Report
title_full_unstemmed Management in Fallot Tetralogy associated with Congenital Scoliosis Case Report
title_short Management in Fallot Tetralogy associated with Congenital Scoliosis Case Report
title_sort management in fallot tetralogy associated with congenital scoliosis case report
topic Original Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6880200/
https://www.ncbi.nlm.nih.gov/pubmed/31803303
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