Cargando…
PRMT1 suppresses ATF4-mediated endoplasmic reticulum response in cardiomyocytes
Endoplasmic reticulum (ER) stress signaling plays a critical role in the control of cell survival or death. Persistent ER stress activates proapoptotic pathway involving the ATF4/CHOP axis. Although accumulating evidences support its important contribution to cardiovascular diseases, but its mechani...
Autores principales: | Jeong, Myong-Ho, Jeong, Hyeon-Ju, Ahn, Byeong-Yun, Pyun, Jung-Hoon, Kwon, Ilmin, Cho, Hana, Kang, Jong-Sun |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2019
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6885520/ https://www.ncbi.nlm.nih.gov/pubmed/31787756 http://dx.doi.org/10.1038/s41419-019-2147-3 |
Ejemplares similares
-
Correction: PRMT1 suppresses ATF4-mediated endoplasmic reticulum response in cardiomyocytes
por: Jeong, Myong-Ho, et al.
Publicado: (2020) -
Cardiac specific PRMT1 ablation causes heart failure through CaMKII dysregulation
por: Pyun, Jung-Hoon, et al.
Publicado: (2018) -
ATF3 regulates SPHK1 in cardiomyocyte injury via endoplasmic reticulum stress
por: Chen, Huiling, et al.
Publicado: (2023) -
Modulation of I
(Ks) channel–PIP(2) interaction by PRMT1 plays a critical role in the control of cardiac repolarization
por: An, Xue, et al.
Publicado: (2022) -
PRMT7 deficiency causes dysregulation of the HCN channels in the CA1 pyramidal cells and impairment of social behaviors
por: Lee, Seul-Yi, et al.
Publicado: (2020)