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Extrachromosomal Histone H2B Contributes to the Formation of the Abscission Site for Cell Division

Histones are constitutive components of nucleosomes and key regulators of chromatin structure. We previously observed that an extrachromosomal histone H2B (ecH2B) localizes at the intercellular bridge (ICB) connecting the two daughter cells during cytokinesis independently of DNA and RNA. Here, we s...

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Autores principales: Monteonofrio, Laura, Valente, Davide, Rinaldo, Cinzia, Soddu, Silvia
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2019
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6912571/
https://www.ncbi.nlm.nih.gov/pubmed/31694230
http://dx.doi.org/10.3390/cells8111391
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author Monteonofrio, Laura
Valente, Davide
Rinaldo, Cinzia
Soddu, Silvia
author_facet Monteonofrio, Laura
Valente, Davide
Rinaldo, Cinzia
Soddu, Silvia
author_sort Monteonofrio, Laura
collection PubMed
description Histones are constitutive components of nucleosomes and key regulators of chromatin structure. We previously observed that an extrachromosomal histone H2B (ecH2B) localizes at the intercellular bridge (ICB) connecting the two daughter cells during cytokinesis independently of DNA and RNA. Here, we show that ecH2B binds and colocalizes with CHMP4B, a key component of the ESCRT-III machinery responsible for abscission, the final step of cell division. Abscission requires the formation of an abscission site at the ICB where the ESCRT-III complex organizes into narrowing cortical helices that drive the physical separation of sibling cells. ecH2B depletion does not prevent membrane cleavage rather results in abscission delay and accumulation of abnormally long and thin ICBs. In the absence of ecH2B, CHMP4B and other components of the fission machinery, such as IST1 and Spastin, are recruited to the ICB and localize at the midbody. However, in the late stage of abscission, these fission factors fail to re-localize at the periphery of the midbody and the abscission site fails to form. These results show that extrachromosomal activity of histone H2B is required in the formation of the abscission site and the proper localization of the fission machinery.
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spelling pubmed-69125712020-01-02 Extrachromosomal Histone H2B Contributes to the Formation of the Abscission Site for Cell Division Monteonofrio, Laura Valente, Davide Rinaldo, Cinzia Soddu, Silvia Cells Article Histones are constitutive components of nucleosomes and key regulators of chromatin structure. We previously observed that an extrachromosomal histone H2B (ecH2B) localizes at the intercellular bridge (ICB) connecting the two daughter cells during cytokinesis independently of DNA and RNA. Here, we show that ecH2B binds and colocalizes with CHMP4B, a key component of the ESCRT-III machinery responsible for abscission, the final step of cell division. Abscission requires the formation of an abscission site at the ICB where the ESCRT-III complex organizes into narrowing cortical helices that drive the physical separation of sibling cells. ecH2B depletion does not prevent membrane cleavage rather results in abscission delay and accumulation of abnormally long and thin ICBs. In the absence of ecH2B, CHMP4B and other components of the fission machinery, such as IST1 and Spastin, are recruited to the ICB and localize at the midbody. However, in the late stage of abscission, these fission factors fail to re-localize at the periphery of the midbody and the abscission site fails to form. These results show that extrachromosomal activity of histone H2B is required in the formation of the abscission site and the proper localization of the fission machinery. MDPI 2019-11-05 /pmc/articles/PMC6912571/ /pubmed/31694230 http://dx.doi.org/10.3390/cells8111391 Text en © 2019 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).
spellingShingle Article
Monteonofrio, Laura
Valente, Davide
Rinaldo, Cinzia
Soddu, Silvia
Extrachromosomal Histone H2B Contributes to the Formation of the Abscission Site for Cell Division
title Extrachromosomal Histone H2B Contributes to the Formation of the Abscission Site for Cell Division
title_full Extrachromosomal Histone H2B Contributes to the Formation of the Abscission Site for Cell Division
title_fullStr Extrachromosomal Histone H2B Contributes to the Formation of the Abscission Site for Cell Division
title_full_unstemmed Extrachromosomal Histone H2B Contributes to the Formation of the Abscission Site for Cell Division
title_short Extrachromosomal Histone H2B Contributes to the Formation of the Abscission Site for Cell Division
title_sort extrachromosomal histone h2b contributes to the formation of the abscission site for cell division
topic Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6912571/
https://www.ncbi.nlm.nih.gov/pubmed/31694230
http://dx.doi.org/10.3390/cells8111391
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