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Extrachromosomal Histone H2B Contributes to the Formation of the Abscission Site for Cell Division
Histones are constitutive components of nucleosomes and key regulators of chromatin structure. We previously observed that an extrachromosomal histone H2B (ecH2B) localizes at the intercellular bridge (ICB) connecting the two daughter cells during cytokinesis independently of DNA and RNA. Here, we s...
Autores principales: | , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2019
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6912571/ https://www.ncbi.nlm.nih.gov/pubmed/31694230 http://dx.doi.org/10.3390/cells8111391 |
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author | Monteonofrio, Laura Valente, Davide Rinaldo, Cinzia Soddu, Silvia |
author_facet | Monteonofrio, Laura Valente, Davide Rinaldo, Cinzia Soddu, Silvia |
author_sort | Monteonofrio, Laura |
collection | PubMed |
description | Histones are constitutive components of nucleosomes and key regulators of chromatin structure. We previously observed that an extrachromosomal histone H2B (ecH2B) localizes at the intercellular bridge (ICB) connecting the two daughter cells during cytokinesis independently of DNA and RNA. Here, we show that ecH2B binds and colocalizes with CHMP4B, a key component of the ESCRT-III machinery responsible for abscission, the final step of cell division. Abscission requires the formation of an abscission site at the ICB where the ESCRT-III complex organizes into narrowing cortical helices that drive the physical separation of sibling cells. ecH2B depletion does not prevent membrane cleavage rather results in abscission delay and accumulation of abnormally long and thin ICBs. In the absence of ecH2B, CHMP4B and other components of the fission machinery, such as IST1 and Spastin, are recruited to the ICB and localize at the midbody. However, in the late stage of abscission, these fission factors fail to re-localize at the periphery of the midbody and the abscission site fails to form. These results show that extrachromosomal activity of histone H2B is required in the formation of the abscission site and the proper localization of the fission machinery. |
format | Online Article Text |
id | pubmed-6912571 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2019 |
publisher | MDPI |
record_format | MEDLINE/PubMed |
spelling | pubmed-69125712020-01-02 Extrachromosomal Histone H2B Contributes to the Formation of the Abscission Site for Cell Division Monteonofrio, Laura Valente, Davide Rinaldo, Cinzia Soddu, Silvia Cells Article Histones are constitutive components of nucleosomes and key regulators of chromatin structure. We previously observed that an extrachromosomal histone H2B (ecH2B) localizes at the intercellular bridge (ICB) connecting the two daughter cells during cytokinesis independently of DNA and RNA. Here, we show that ecH2B binds and colocalizes with CHMP4B, a key component of the ESCRT-III machinery responsible for abscission, the final step of cell division. Abscission requires the formation of an abscission site at the ICB where the ESCRT-III complex organizes into narrowing cortical helices that drive the physical separation of sibling cells. ecH2B depletion does not prevent membrane cleavage rather results in abscission delay and accumulation of abnormally long and thin ICBs. In the absence of ecH2B, CHMP4B and other components of the fission machinery, such as IST1 and Spastin, are recruited to the ICB and localize at the midbody. However, in the late stage of abscission, these fission factors fail to re-localize at the periphery of the midbody and the abscission site fails to form. These results show that extrachromosomal activity of histone H2B is required in the formation of the abscission site and the proper localization of the fission machinery. MDPI 2019-11-05 /pmc/articles/PMC6912571/ /pubmed/31694230 http://dx.doi.org/10.3390/cells8111391 Text en © 2019 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/). |
spellingShingle | Article Monteonofrio, Laura Valente, Davide Rinaldo, Cinzia Soddu, Silvia Extrachromosomal Histone H2B Contributes to the Formation of the Abscission Site for Cell Division |
title | Extrachromosomal Histone H2B Contributes to the Formation of the Abscission Site for Cell Division |
title_full | Extrachromosomal Histone H2B Contributes to the Formation of the Abscission Site for Cell Division |
title_fullStr | Extrachromosomal Histone H2B Contributes to the Formation of the Abscission Site for Cell Division |
title_full_unstemmed | Extrachromosomal Histone H2B Contributes to the Formation of the Abscission Site for Cell Division |
title_short | Extrachromosomal Histone H2B Contributes to the Formation of the Abscission Site for Cell Division |
title_sort | extrachromosomal histone h2b contributes to the formation of the abscission site for cell division |
topic | Article |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6912571/ https://www.ncbi.nlm.nih.gov/pubmed/31694230 http://dx.doi.org/10.3390/cells8111391 |
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