Cargando…
Mild Traumatic Brain Injury Causes Nociceptive Sensitization through Spinal Chemokine Upregulation
High rates of acute and chronic pain are associated with traumatic brain injury (TBI), but mechanisms responsible for the association remain elusive. Recent data suggest dysregulated descending pain modulation circuitry could be involved. Based on these and other observations, we hypothesized that s...
Autores principales: | Sahbaie, Peyman, Irvine, Karen-Amanda, Liang, De-Yong, Shi, Xiaoyou, Clark, J. David |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2019
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6925232/ https://www.ncbi.nlm.nih.gov/pubmed/31863005 http://dx.doi.org/10.1038/s41598-019-55739-x |
Ejemplares similares
-
The chemokine receptor CXCR2 supports nociceptive sensitization after traumatic brain injury
por: Liang, De-Yong, et al.
Publicado: (2017) -
Monoamine control of descending pain modulation after mild traumatic brain injury
por: Sahbaie, Peyman, et al.
Publicado: (2022) -
TBI-induced nociceptive sensitization is regulated by histone acetylation
por: Liang, De-Yong, et al.
Publicado: (2016) -
DNA Methylation Modulates Nociceptive Sensitization after Incision
por: Sun, Yuan, et al.
Publicado: (2015) -
Facilitated spinal neuropeptide signaling and upregulated inflammatory mediator expression contribute to postfracture nociceptive sensitization
por: Shi, Xiaoyou, et al.
Publicado: (2015)