Cargando…
The expression of miR‐125b in Nrf2‐silenced A549 cells exposed to hyperoxia and its relationship with apoptosis
Bronchopulmonary dysplasia (BPD) is a chronic lung disease that affects the quality of life of infants. At present, premature exposure to hyperoxia for extended periods of time is believed to affect the development of lung tissue and vascularity, resulting in BPD. The oxidative stress caused by hype...
Autores principales: | Zhang, Xiaoyue, Chu, Xiaoyun, Gong, Xiaohui, Zhou, Huilin, Cai, Cheng |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2019
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6933325/ https://www.ncbi.nlm.nih.gov/pubmed/31713992 http://dx.doi.org/10.1111/jcmm.14808 |
Ejemplares similares
-
NRF2 Alters Mitochondrial Gene Expression in Neonate Mice Exposed to Hyperoxia
por: Vellers, Heather L., et al.
Publicado: (2022) -
Hypermethylation-Associated Silencing of miR-125a and miR-125b: A Potential Marker in Colorectal Cancer
por: Chen, Hui, et al.
Publicado: (2015) -
BMI1 Silencing Induces Mitochondrial Dysfunction in Lung Epithelial Cells Exposed to Hyperoxia
por: Hernández-Cuervo, Helena, et al.
Publicado: (2022) -
Caffeine induces alveolar apoptosis in the hyperoxia-exposed developing mouse lung
por: Dayanim, Sara, et al.
Publicado: (2013) -
Long non-coding RNA MALAT1 plays a protective role in bronchopulmonary dysplasia via the inhibition of apoptosis and interaction with the Keap1/Nrf2 signal pathway
por: Zhang, Min, et al.
Publicado: (2021)