Cargando…
Annexin A2 Deficiency Exacerbates Neuroinflammation and Long-Term Neurological Deficits after Traumatic Brain Injury in Mice
Our laboratory and others previously showed that Annexin A2 knockout (A2KO) mice had impaired blood–brain barrier (BBB) development and elevated pro-inflammatory response in macrophages, implying that Annexin A2 (AnxA2) might be one of the key endogenous factors for maintaining homeostasis of the ne...
Autores principales: | Liu, Ning, Jiang, Yinghua, Chung, Joon Yong, Li, Yadan, Yu, Zhanyang, Kim, Jeong Woo, Lok, Josephine M., Whalen, Michael J., Wang, Xiaoying |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2019
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6940735/ https://www.ncbi.nlm.nih.gov/pubmed/31817350 http://dx.doi.org/10.3390/ijms20246125 |
Ejemplares similares
-
Recombinant Annexin A2 Administration Improves Neurological Outcomes After Traumatic Brain Injury in Mice
por: Cheng, Chongjie, et al.
Publicado: (2021) -
Recombinant annexin A2 inhibits peripheral leukocyte activation and brain infiltration after traumatic brain injury
por: Liu, Ning, et al.
Publicado: (2021) -
Thrombospondin-1 Gene Deficiency Worsens the Neurological Outcomes of Traumatic Brain Injury in Mice
por: Cheng, Chongjie, et al.
Publicado: (2017) -
Post-traumatic hypoxia exacerbates neurological deficit, neuroinflammation and cerebral metabolism in rats with diffuse traumatic brain injury
por: Yan, Edwin B, et al.
Publicado: (2011) -
Neuroglobin-overexpression reduces traumatic brain lesion size in mice
por: Zhao, Song, et al.
Publicado: (2012)