Cargando…
Author Correction: Dysfunctional EAT thickness may promote maladaptive heart remodeling in CVD patients through the ST2-IL33 system, directly related to EPAC protein expression
Autores principales: | Vianello, Elena, Dozio, Elena, Bandera, Francesco, Schmitz, Gerd, Nebuloni, Manuela, Longhi, Erika, Tacchini, Lorenza, Guazzi, Marco, Corsi Romanelli, Massimiliano Marco |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2020
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6970986/ https://www.ncbi.nlm.nih.gov/pubmed/31959862 http://dx.doi.org/10.1038/s41598-020-58143-y |
Ejemplares similares
-
Dysfunctional EAT thickness may promote maladaptive heart remodeling in CVD patients through the ST2-IL33 system, directly related to EPAC protein expression
por: Vianello, Elena, et al.
Publicado: (2019) -
Correlative Study on Impaired Prostaglandin E2 Regulation in Epicardial Adipose Tissue and Its Role in Maladaptive Cardiac Remodeling via EPAC2 and ST2 Signaling in Overweight Cardiovascular Disease Subjects
por: Vianello, Elena, et al.
Publicado: (2020) -
Expression of the Receptor for Advanced Glycation End Products in Epicardial Fat: Link with Tissue Thickness and Local Insulin Resistance in Coronary Artery Disease
por: Dozio, Elena, et al.
Publicado: (2016) -
Soluble Receptor for Advanced Glycation End Products: A Protective Molecule against Intramyocardial Lipid Accumulation in Obese Zucker Rats?
por: Dozio, Elena, et al.
Publicado: (2019) -
PCSK9 Expression in Epicardial Adipose Tissue: Molecular Association with Local Tissue Inflammation
por: Dozio, Elena, et al.
Publicado: (2020)