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Expression of Concern to: Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatoxicity

The Editors-in-Chief would like to alert readers that this article [1] is part of an investigation being conducted by the journal following the conclusions of an institutional enquiry at the University of Liverpool with respect to the quantitative mass spectrometry-generated results regarding acetyl...

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Autores principales: Antoine, Daniel James, Williams, Dominic P., Kipar, Anja, Laverty, Hugh, Park, B. Kevin
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2020
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6993424/
https://www.ncbi.nlm.nih.gov/pubmed/32000658
http://dx.doi.org/10.1186/s10020-020-0140-z
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author Antoine, Daniel James
Williams, Dominic P.
Kipar, Anja
Laverty, Hugh
Park, B. Kevin
author_facet Antoine, Daniel James
Williams, Dominic P.
Kipar, Anja
Laverty, Hugh
Park, B. Kevin
author_sort Antoine, Daniel James
collection PubMed
description The Editors-in-Chief would like to alert readers that this article [1] is part of an investigation being conducted by the journal following the conclusions of an institutional enquiry at the University of Liverpool with respect to the quantitative mass spectrometry-generated results regarding acetylated and redox-modified HMGB1.
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spelling pubmed-69934242020-02-04 Expression of Concern to: Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatoxicity Antoine, Daniel James Williams, Dominic P. Kipar, Anja Laverty, Hugh Park, B. Kevin Mol Med Expression of Concern The Editors-in-Chief would like to alert readers that this article [1] is part of an investigation being conducted by the journal following the conclusions of an institutional enquiry at the University of Liverpool with respect to the quantitative mass spectrometry-generated results regarding acetylated and redox-modified HMGB1. BioMed Central 2020-01-30 /pmc/articles/PMC6993424/ /pubmed/32000658 http://dx.doi.org/10.1186/s10020-020-0140-z Text en © The Author(s) 2020 Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.
spellingShingle Expression of Concern
Antoine, Daniel James
Williams, Dominic P.
Kipar, Anja
Laverty, Hugh
Park, B. Kevin
Expression of Concern to: Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatoxicity
title Expression of Concern to: Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatoxicity
title_full Expression of Concern to: Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatoxicity
title_fullStr Expression of Concern to: Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatoxicity
title_full_unstemmed Expression of Concern to: Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatoxicity
title_short Expression of Concern to: Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatoxicity
title_sort expression of concern to: diet restriction inhibits apoptosis and hmgb1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatoxicity
topic Expression of Concern
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6993424/
https://www.ncbi.nlm.nih.gov/pubmed/32000658
http://dx.doi.org/10.1186/s10020-020-0140-z
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