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A role for the rare endogenous retrovirus β4 in development of Japanese fancy mice
Two coat-color mutations, nonagouti, which changes coat color from wild-type agouti to black, and piebald, which induces irregular white spotting, are the characteristics of Japanese fancy mouse strain JF1/Ms. In our Communications Biology article, we reported that insertion of a rare type of endoge...
Autores principales: | , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2020
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7000388/ https://www.ncbi.nlm.nih.gov/pubmed/32020010 http://dx.doi.org/10.1038/s42003-020-0781-z |
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author | Tanave, Akira Koide, Tsuyoshi |
author_facet | Tanave, Akira Koide, Tsuyoshi |
author_sort | Tanave, Akira |
collection | PubMed |
description | Two coat-color mutations, nonagouti, which changes coat color from wild-type agouti to black, and piebald, which induces irregular white spotting, are the characteristics of Japanese fancy mouse strain JF1/Ms. In our Communications Biology article, we reported that insertion of a rare type of endogenous retrovirus β4 has caused both coat color mutations. Although there are some reports on the roles of β4 in the mouse genome, further studies on β4 will uncover new features of endogenous retrovirus sequences. |
format | Online Article Text |
id | pubmed-7000388 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2020 |
publisher | Nature Publishing Group UK |
record_format | MEDLINE/PubMed |
spelling | pubmed-70003882020-02-11 A role for the rare endogenous retrovirus β4 in development of Japanese fancy mice Tanave, Akira Koide, Tsuyoshi Commun Biol Comment Two coat-color mutations, nonagouti, which changes coat color from wild-type agouti to black, and piebald, which induces irregular white spotting, are the characteristics of Japanese fancy mouse strain JF1/Ms. In our Communications Biology article, we reported that insertion of a rare type of endogenous retrovirus β4 has caused both coat color mutations. Although there are some reports on the roles of β4 in the mouse genome, further studies on β4 will uncover new features of endogenous retrovirus sequences. Nature Publishing Group UK 2020-02-04 /pmc/articles/PMC7000388/ /pubmed/32020010 http://dx.doi.org/10.1038/s42003-020-0781-z Text en © The Author(s) 2020 Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/. |
spellingShingle | Comment Tanave, Akira Koide, Tsuyoshi A role for the rare endogenous retrovirus β4 in development of Japanese fancy mice |
title | A role for the rare endogenous retrovirus β4 in development of Japanese fancy mice |
title_full | A role for the rare endogenous retrovirus β4 in development of Japanese fancy mice |
title_fullStr | A role for the rare endogenous retrovirus β4 in development of Japanese fancy mice |
title_full_unstemmed | A role for the rare endogenous retrovirus β4 in development of Japanese fancy mice |
title_short | A role for the rare endogenous retrovirus β4 in development of Japanese fancy mice |
title_sort | role for the rare endogenous retrovirus β4 in development of japanese fancy mice |
topic | Comment |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7000388/ https://www.ncbi.nlm.nih.gov/pubmed/32020010 http://dx.doi.org/10.1038/s42003-020-0781-z |
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