Cargando…
c-Met is expressed by highly autoreactive encephalitogenic CD8+ cells
BACKGROUND: CD8(+) T lymphocytes are critical mediators of neuroinflammatory diseases. Understanding the mechanisms that govern the function of this T cell population is crucial to better understanding central nervous system autoimmune disease pathology. We recently identified a novel population of...
Autores principales: | Benkhoucha, Mahdia, Senoner, Isis, Lalive, Patrice H. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2020
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7031922/ https://www.ncbi.nlm.nih.gov/pubmed/32075650 http://dx.doi.org/10.1186/s12974-019-1676-0 |
Ejemplares similares
-
c-Met enforces proinflammatory and migratory features of human activated CD4(+) T cells
por: Benkhoucha, Mahdia, et al.
Publicado: (2021) -
CD4(+)c-Met(+)Itgα4(+) T cell subset promotes murine neuroinflammation
por: Benkhoucha, Mahdia, et al.
Publicado: (2022) -
The neurotrophic hepatocyte growth factor attenuates CD8(+) cytotoxic T-lymphocyte activity
por: Benkhoucha, Mahdia, et al.
Publicado: (2013) -
Activation of human B cells negatively regulates TGF-β1 production
por: Molnarfi, Nicolas, et al.
Publicado: (2017) -
Interferon–β Induces Hepatocyte Growth Factor in Monocytes of Multiple Sclerosis Patients
por: Molnarfi, Nicolas, et al.
Publicado: (2012)